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Original Research

Inhibition of gap junction intercellular communication is involved in silica nanoparticles-induced H9c2 cardiomyocytes apoptosis via the mitochondrial pathway

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Pages 2179-2188 | Published online: 20 Mar 2017

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Ya-Qin Liu, Si-Meng Xue, Peng Zhang, Lin-Na Xu, De-Ping Wang, Guang Li & Ji-Min Cao. (2020) Silica Nanoparticles Disturb Ion Channels and Transmembrane Potentials of Cardiomyocytes and Induce Lethal Arrhythmias in Mice. International Journal of Nanomedicine 15, pages 7397-7413.
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Guanqun Cui, Haiyang Zhang, Qiming Guo, Shan Shan, Shangya Chen, Chao Li, Xu Yang, Ziyuan Li, Yingwen Mu, Hua Shao & Zhongjun Du. (2020) Oxidative stress-mediated mitochondrial pathway-dependent apoptosis is induced by silica nanoparticles in H9c2 cardiomyocytes. Toxicology Mechanisms and Methods 30:9, pages 646-655.
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Lin Feng, Xiaozhe Yang, Yanfeng Shi, Shuang Liang, Tong Zhao, Junchao Duan & Zhiwei Sun. (2018) Co-exposure subacute toxicity of silica nanoparticles and lead acetate on cardiovascular system. International Journal of Nanomedicine 13, pages 7819-7834.
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Ling Wang, Chao Chen, Lijie Guo, Qin Li, Hongyan Ding, Hongsheng Bi & Dadong Guo. (2018) Zinc oxide nanoparticles induce murine photoreceptor cell death via mitochondria-related signaling pathway. Artificial Cells, Nanomedicine, and Biotechnology 46:sup1, pages 1102-1113.
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