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Scandinavian Journal of Gastroenterology Volume 18, 1983 - Issue 5
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Original Article

Metabolism of the NH2-Terminal Tridecapeptide of Gastrin-17 in Normal Subjects and Duodenal Ulcer Patients

B. Petersen Dept. of Surgery D, Glostrup Hospital, and University Dept. of Clinical Chemistry, Rigshospitalet, Copenhagen, Denmark
Pages 613-617 | Received 22 Sep 1982, Accepted 02 Nov 1982, Published online: 19 Feb 2010

Citations (7)

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Read on this site (2)

B. Petersen. (1984) Abnormally processed gastrins in active duodenal ulcer disease. Scandinavian Journal of Clinical and Laboratory Investigation 44:sup168, pages 25-44.
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B. Petersen, J. Steen & J. Christiansen. (1983) Variations in the Release of the NH2-Terminal Fragment of Gastrin-17 in the Course of Duodenal Ulcer Disease. Scandinavian Journal of Gastroenterology 18:8, pages 1101-1107.
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Articles from other publishers (5)

Linda Hilsted, Kathrin Hint, John Christiansen & Jens F. Rehfeld. (1988) Neither glycine-extended gastrin nor the 1–13 fragment of gastrin 17 influences gastric acid secretion in humans. Gastroenterology 94:1, pages 96-102.
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Piyush C. Kothary & Aaron I. Vinik. (1987) NH2-Terminal of gastrin-17 in duodenal ulcer disease: Identification of progastrin-17. Biochemical and Biophysical Research Communications 146:2, pages 884-888.
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S. Pauwels, G.J. Dockray, R. Walker & S. Marcus. (1985) Metabolism of the 1–13 sequence of gastrin 17 in humans and failure to influence acid secretion. Gastroenterology 89:1, pages 49-56.
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BIRTE PETERSEN & BENT NYBOE ANDERSEN. (2008) Abnormal processing of antral gastrin in active duodenal ulcer disease. European Journal of Clinical Investigation 14:3, pages 214-218.
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Birte Petersen, John Christiansen & Jens F. Rehfeld. (1983) The N-terminal tridecapeptide fragment of gastrin-17 inhibits gastric acid secretion. Regulatory Peptides 7:4, pages 323-334.
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