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Hemoglobin
international journal for hemoglobin research
Volume 23, 1999 - Issue 2
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Original Article

The β+-thalassemia mutation [IVS-II-5 (G→C)] creates an alternative splicing site in the second intervening sequence

N. H. Jiang Department of Pediatrics, First Hospital of Guangxi Medical University Nanning, Guangxi, People's Republic of ChinaCorrespondence[email protected]
&
S. Liang Department of Pediatrics, First Hospital of Guangxi Medical University Nanning, Guangxi, People's Republic of China
Pages 171-176 | Received 31 Jul 1998, Accepted 09 Nov 1998, Published online: 05 Aug 2009

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Yuling Qiu, Shilu Wei, Wei Hou, Ketong Lai, Hengying Zhu, Wenwei Li, Qi Li, Zheng Yang, Qisheng Shu, Ping Chen & Wuning Mo. (2023) Molecular Basis and Hematologic Phenotype of Hemoglobin H Disease Combined with Two Rare β-Globin Mutations. Hemoglobin 47:2, pages 52-55.
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Lin Zhao, Jilin Qing, Yue Liang & Zhizhong Chen. (2016) A novel compound heterozygosity in Southern China: IVS-II-5 (G > C) and IVS-II-672 (A > C). Hemoglobin 40:6, pages 428-430.
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Articles from other publishers (1)

Petros Patsali, Claudio Mussolino, Petros Ladas, Argyro Floga, Annita Kolnagou, Soteroula Christou, Maria Sitarou, Michael N. Antoniou, Toni Cathomen, Carsten Werner Lederer & Marina Kleanthous. (2019) The Scope for Thalassemia Gene Therapy by Disruption of Aberrant Regulatory Elements. Journal of Clinical Medicine 8:11, pages 1959.
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