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Cancer Investigation Volume 28, 2010 - Issue 1
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LETTER TO THE EDITOR

Bevacizumab may be Active in Myelofibrosis

Ilias NikolaidisOncology Unit, 3rd Department of Medicine, Sotiria General Hospital, Athens School of Medicine, Greece, 1st Department of Medicine, Laiko General Hospital, Athens School of Medicine, Greece.
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Nektaria MakriliaOncology Unit, 3rd Department of Medicine, Sotiria General Hospital, Athens School of Medicine, Greece, 1st Department of Medicine, Laiko General Hospital, Athens School of Medicine, Greece.
,
Vassilia XylaOncology Unit, 3rd Department of Medicine, Sotiria General Hospital, Athens School of Medicine, Greece, 1st Department of Medicine, Laiko General Hospital, Athens School of Medicine, Greece.
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Kalliopi DilanaOncology Unit, 3rd Department of Medicine, Sotiria General Hospital, Athens School of Medicine, Greece, 1st Department of Medicine, Laiko General Hospital, Athens School of Medicine, Greece.
,
Dimitris StathopoulosOncology Unit, 3rd Department of Medicine, Sotiria General Hospital, Athens School of Medicine, Greece, 1st Department of Medicine, Laiko General Hospital, Athens School of Medicine, Greece.
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Ioannis MeletisOncology Unit, 3rd Department of Medicine, Sotiria General Hospital, Athens School of Medicine, Greece, 1st Department of Medicine, Laiko General Hospital, Athens School of Medicine, Greece.
&
Kostas SyrigosOncology Unit, 3rd Department of Medicine, Sotiria General Hospital, Athens School of Medicine, Greece, 1st Department of Medicine, Laiko General Hospital, Athens School of Medicine, Greece.Correspondence[email protected] [email protected]
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Pages 111-112 | Published online: 09 Dec 2009

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Claire N Harrison & Michael Bennett. (2014) Orphan drugs for myelofibrosis. Expert Opinion on Orphan Drugs 2:4, pages 391-405.
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Jacek Treliński, Agnieszka Wierzbowska, Anna Krawczyńska, Agata Sakowicz, Tadeusz Pietrucha, Piotr Smolewski, Tadeusz Robak & Krzysztof Chojnowski. (2010) Plasma levels of angiogenic factors and circulating endothelial cells in essential thrombocythemia: correlation with cytoreductive therapy and JAK2–V617F mutational status. Leukemia & Lymphoma 51:9, pages 1-7.
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