IL-35 Is Involved in the Pathogenesis of Guillain–Barré Syndrome Through Its Influence on the Function of CD4+ T Cells

Hao LiuDepartment of Neurology, Xiangya Hospital, Central South University, Changsha, Hunan, PR China,

Wei ZhangDepartment of Neurology, Central Hospital of Xiangtan, Xiangtan, Hunan, PR China, and

Fa-Fa TianDepartment of Neurology, Xiangya Hospital, Central South University, Changsha, Hunan, PR China,

An KunDepartment of Neurology, Huai’an First People’s Hospital, Huai’an, Jiangsu, PR China

Wen-Bin ZhouDepartment of Neurology, Xiangya Hospital, Central South University, Changsha, Hunan, PR China, Correspondence[email protected]

Bo XiaoDepartment of Neurology, Xiangya Hospital, Central South University, Changsha, Hunan, PR China,

Jing LiDepartment of Neurology, Xiangya Hospital, Central South University, Changsha, Hunan, PR China,

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IL-35 Is Involved in the Pathogenesis of Guillain–Barré Syndrome Through Its Influence on the Function of CD4+ T Cells

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IL-35 Is Involved in the Pathogenesis of Guillain–Barré Syndrome Through Its Influence on the Function of CD4+ T Cells

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