A Herpes simplex virus type 1 mutant with a deletion immediately upstream of the LAT locus establishes latency and reactivates from latently infected mice with normal kinetics

John Maggioncalda Jefferson Cancer Institute, Thomas Jefferson University, 1020 Locust Street, Philadelphia, Pennsylvania, 19107-6799, USA

Anand Mehta Jefferson Cancer Institute, Thomas Jefferson University, 1020 Locust Street, Philadelphia, Pennsylvania, 19107-6799, USA

Omar Bagasra Department of Medicine, Thomas Jefferson University, 1020 Locust Street, Philadelphia, Pennsylvania, 19107-6799, USA

Nigel W Fraser The Wistar Institute, 36th and Spruce Streets, Philadelphia, Pennsylvania, 19101, USA

Timothy M Block Jefferson Cancer Institute, Thomas Jefferson University, 1020 Locust Street, Philadelphia, Pennsylvania, 19107-6799, USA; The Wistar Institute, 36th and Spruce Streets, Philadelphia, Pennsylvania, 19101, USA

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Ling Jin, Guey-Chuen Perng, Dale Carpenter, Kevin R Mott, Nelson Osorio, Julia Naito, David J Brick, Clinton Jones & Steven L Wechsler. (2007) Reactivation phenotype in rabbits of a herpes simplex virus type 1 mutant containing an unrelated antiapoptosis gene in place of latency-associated transcript. Journal of Neurovirology 13:1, pages 78-84.
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David Chan, Jacob Cohen, Julie Naito, Kevin R Mott, Nelson Osorio, Ling Jin, Nigel W Fraser, Clinton Jones, Steven L Wechsler & Guey Chuen Perng. (2006) A mutant deleted for most of the herpes simplex virus type 1 (HSV-1) UOL gene does not affect the spontaneous reactivation phenotype in rabbits. Journal of Neurovirology 12:1, pages 5-16.
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A Herpes simplex virus type 1 mutant with a deletion immediately upstream of the LAT locus establishes latency and reactivates from latently infected mice with normal kinetics

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A Herpes simplex virus type 1 mutant with a deletion immediately upstream of the LAT locus establishes latency and reactivates from latently infected mice with normal kinetics

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