Impaired NFKBIE gene function decreases cellular uptake of methotrexate by down-regulating SLC19A1 expression in a human rheumatoid arthritis cell line

Hitoshi ImamuraDepartment of Physiology, Tokyo Women’s Medical University School of Medicine, Tokyo, Japan, ;Institute of Rheumatology, Tokyo Women's Medical University, Tokyo, Japan,

Sawako YoshinaDepartment of Physiology, Tokyo Women’s Medical University School of Medicine, Tokyo, Japan,

Katsunori IkariInstitute of Rheumatology, Tokyo Women's Medical University, Tokyo, Japan,

Keiji MiyazawaDiscovery Research III, Research and Development, Kissei Pharmaceutical Company, Nagano, Japan, and

Shigeki MomoharaInstitute of Rheumatology, Tokyo Women's Medical University, Tokyo, Japan,

Shohei MitaniDepartment of Physiology, Tokyo Women’s Medical University School of Medicine, Tokyo, Japan, ;Tokyo Women’s Medical University Institute for Integrated Medical Sciences, Tokyo, JapanCorrespondence[email protected]

Impaired NFKBIE gene function decreases cellular uptake of methotrexate by down-regulating SLC19A1 expression in a human rheumatoid arthritis cell line

Articles from other publishers (8)

Information for

Open access

Opportunities

Help and information

Your download is now in progress and you may close this window

Login or register to access this feature

Impaired NFKBIE gene function decreases cellular uptake of methotrexate by down-regulating SLC19A1 expression in a human rheumatoid arthritis cell line

Citations (8)

Articles from other publishers (8)

Related research

To cite this article:

Download citation

Information for

Open access

Opportunities

Help and information

Keep up to date