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Research Article

Senescence in amniocytes and placentas from trisomy 21 pregnancies

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Pages 1086-1089 | Received 12 Nov 2012, Accepted 15 Jan 2013, Published online: 20 Feb 2013

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Karampreet Kaur, Ganesh Acharya, Heidi Chen, Chevis N. Shannon, Brittany E. Lipscomb, Randa Newman & Lisa C. Zuckerwise. (2022) Impact of fetal trisomy 21 on umbilical artery Doppler indices. The Journal of Maternal-Fetal & Neonatal Medicine 35:25, pages 8364-8371.
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Articles from other publishers (16)

Guang-Shun Gong, Kahindo P Muyayalo, Yu-Jing Zhang, Xin-Xiu Lin & Ai-Hua Liao. (2023) Flip a coin: cell senescence at the maternal–fetal interface. Biology of Reproduction 109:3, pages 244-255.
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Meijun Pan, Jing Zhou, Jing Wang, Wenli Cao, Lisha Li & Ling Wang. (2023) The role of placental aging in adverse pregnancy outcomes: A mitochondrial perspective. Life Sciences 329, pages 121924.
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April D. Adams, Jielu Lin, Diana W. Bianchi, Lauren Bishop, Taisuke Sato, Laura L. Baxter, Victoria Hoffmann, Laura Koehly & Faycal Guedj. (2023) Embryonic statistical analyses reveal 2 growth phenotypes in mouse models of Down syndrome. American Journal of Obstetrics and Gynecology.
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Daniel Stott, Francois Dos Santos, Anna Rodgers, Elaine Holgado & Pranav P. Pandya. (2022) Antenatal findings and early postnatal outcomes in pregnancies with trisomy 21: a 10 year retrospective review at a tertiary centre. Prenatal Diagnosis 42:10, pages 1273-1280.
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Manas Ranjan Sahu, Linchi Rani, Rhea Subba & Amal Chandra Mondal. (2022) Cellular senescence in the aging brain: A promising target for neurodegenerative diseases. Mechanisms of Ageing and Development 204, pages 111675.
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Noemí Rueda Revilla & Carmen Martínez-Cué. (2020) Antioxidants in Down Syndrome: From Preclinical Studies to Clinical Trials. Antioxidants 9:8, pages 692.
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Carmen Martínez-Cué & Noemí Rueda. (2020) Cellular Senescence in Neurodegenerative Diseases. Frontiers in Cellular Neuroscience 14.
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Sarah E. Lee, Monica Duran-Martinez, Sabina Khantsis, Diana W. Bianchi & Faycal Guedj. (2020) Challenges and Opportunities for Translation of Therapies to Improve Cognition in Down Syndrome. Trends in Molecular Medicine 26:2, pages 150-169.
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April D. Adams, Faycal Guedj & Diana W. Bianchi. (2020) Placental development and function in trisomy 21 and mouse models of Down syndrome: Clues for studying mechanisms underlying atypical development. Placenta 89, pages 58-66.
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Claudio Franceschi, Paolo Garagnani, Noémie Gensous, Maria Giulia Bacalini, Maria Conte & Stefano Salvioli. (2019) Accelerated bio‐cognitive aging in Down syndrome: State of the art and possible deceleration strategies. Aging Cell 18:3.
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Luis Ángel Maciel-BarónDaniel Moreno-BlasSandra Lizbeth Morales-RosalesViridiana Yazmín González-PuertosNorma Edith López-DíazguerreroClaudio TorresSusana Castro-ObregónMina Königsberg. (2018) Cellular Senescence, Neurological Function, and Redox State. Antioxidants & Redox Signaling 28:18, pages 1704-1723.
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Lynne S. Cox & Christopher Redman. (2017) The role of cellular senescence in ageing of the placenta. Placenta 52, pages 139-145.
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Faycal Guedj, Jeroen LA Pennings, Lauren J. Massingham, Heather C. Wick, Ashley E. Siegel, Umadevi Tantravahi & Diana W. Bianchi. (2016) An Integrated Human/Murine Transcriptome and Pathway Approach To Identify Prenatal Treatments For Down Syndrome. Scientific Reports 6:1.
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J. Polettini, E.H. Dutta, F. Behnia, G.R. Saade, M.R. Torloni & R. Menon. (2015) Aging of intrauterine tissues in spontaneous preterm birth and preterm premature rupture of the membranes: A systematic review of the literature. Placenta 36:9, pages 969-973.
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Tal Biron-Shental, Meital Liberman, Merav Sharvit, Rivka Sukenik-Halevy & Aliza Amiel. (2015) Amniocytes from aneuploidy embryos have enhanced random aneuploidy and signs of senescence — Can these findings be related to medical problems?. Gene 562:2, pages 232-235.
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Ido Laish, Hila Katz, Yael Sulayev, Meytal Liberman, Timna Naftali, Fabiana Benjaminov, Assaf Stein, Yona Kitay-Cohen, Tal Biron-Shental, Fred Konikoff & Aliza Amiel. (2013) Increased TERC gene copy number and cells in senescence in primary sclerosing cholangitis compared to colitis and control patients. Gene 529:2, pages 245-249.
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