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Cell Cycle Volume 13, 2014 - Issue 14
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Silencing of Dicer1 temporally separates pro- and anti-apoptotic signaling and confers susceptibility to chemotherapy in p53 mutated cells

Tatyana S Nekova Department of Internal Medicine II; Julius-Maximilians University; Wuerzburg, GermanyCorrespondence[email protected]
,
Susanne Kneitz Physiological Chemistry I; Biocenter; Julius-Maximilians University; Wuerzburg, Germany
,
Hermann Einsele Department of Internal Medicine II; Julius-Maximilians University; Wuerzburg, Germany
&
Gernot Stuhler Department of Internal Medicine II; Julius-Maximilians University; Wuerzburg, Germany
Pages 2192-2198 | Received 02 May 2014, Accepted 12 May 2014, Published online: 20 May 2014

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Tatyana S. Nekova, Susanne Kneitz, Hermann Einsele, Ralf Bargou & Gernot Stuhler. (2016) Silencing of CDK2, but not CDK1, separates mitogenic from anti-apoptotic signaling, sensitizing p53 defective cells for synthetic lethality. Cell Cycle 15:23, pages 3203-3209.
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Emanuele Canestrari & Zain Paroo. (2018) Ribonucleases as Drug Targets. Trends in Pharmacological Sciences 39:10, pages 855-866.
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