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Perspectives

H2AX May Function as an Anchor to Hold Broken Chromosomal DNA Ends in Close Proximity

Pages 147-148 | Published online: 03 Dec 2003

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Esmeralda C. Teo, Benigno C. Valdez, Jie Ji, Yang Li, Yan Liu, Jonathan E. Brammer, Chitra Hosing, Yago Nieto, Richard E. Champlin & Borje S. Andersson. (2016) Synergistic cytotoxicity of busulfan, melphalan, gemcitabine, panobinostat, and bortezomib in lymphoma cells. Leukemia & Lymphoma 57:11, pages 2644-2652.
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Pan Kyu Park, Dong Hyun Kang & Hyockman Kwon. (2009) BAF53 is critical for focus formation of γ‐H2AX in response to DNA damage. Animal Cells and Systems 13:4, pages 405-409.
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M. S. Sasaki. (2009) Advances in the biophysical and molecular bases of radiation cytogenetics. International Journal of Radiation Biology 85:1, pages 26-47.
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V. Ashutosh Rao, Angela M. Fan, LingHua Meng, Christopher F. Doe, Phillip S. North, Ian D. Hickson & Yves Pommier. (2005) Phosphorylation of BLM, Dissociation from Topoisomerase IIIα, and Colocalization with γ-H2AX after Topoisomerase I-Induced Replication Damage. Molecular and Cellular Biology 25:20, pages 8925-8937.
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Toru M. Nakamura, Li-Lin Du, Christophe Redon & Paul Russell. (2004) Histone H2A Phosphorylation Controls Crb2 Recruitment at DNA Breaks, Maintains Checkpoint Arrest, and Influences DNA Repair in Fission Yeast. Molecular and Cellular Biology 24:14, pages 6215-6230.
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