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Fetal and Pediatric Pathology Volume 38, 2019 - Issue 2
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Case Reports

Giant Hepatic Hemangioma and Placental Chorangiosis: A Unique Case of Stillbirth?

Michele PaudicePathology, DISC, University of Genoa, Genoa, Italy; Correspondence[email protected]
,
Leonardo Alett PeñuelaFetal and Placental Pathology Unit, G. Gaslini Institute, Genoa, Italy;
,
Flaminia TorielliNeonatology Unit, University of Genoa, Azienda Ospedaliera San Martino IRCCS – IST National Institute on Cancer Research, Genoa, Italy;
,
Bruno SpinaPathology Unit, San Martino Hospital, Genoa, Italy;
,
Valentino RemorgidaDepartment of Neurosciences, Rehabilitation, Ophthalmology, Genetics, Maternal and Child Health, University of Genoa, Genoa, Italy
,
Francesca BuffelliFetal and Placental Pathology Unit, G. Gaslini Institute, Genoa, Italy;
,
Ezio FulcheriPathology, DISC, University of Genoa, Genoa, Italy; ;Fetal and Placental Pathology Unit, G. Gaslini Institute, Genoa, Italy;
,
Cesare ArioniNeonatology Unit, University of Genoa, Azienda Ospedaliera San Martino IRCCS – IST National Institute on Cancer Research, Genoa, Italy;
&
Valerio Gaetano VellonePathology, DISC, University of Genoa, Genoa, Italy;
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Pages 175-181 | Received 29 Oct 2018, Accepted 11 Dec 2018, Published online: 24 Jan 2019
 
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Abstract

Introduction: Hepatic hemangiomas (HH) are benign vascular tumors, and when large, may contribute to fetal morbidity/mortality. Chorangiosis is placental villus capillary hypervascularity, probably linked with fetal hypoxia. Case report: We present a macrosomic stillbirth at 39 + 3 weeks of gestation with congestive heart failure (CHF) and myocardial infarction. A giant right hepatic lobe HH was present, along with placental chorangiosis. Conclusion: A common pathogenetic pathway between congenital HH and placental chorangiosis has not been reported. Our case suggests that the effects of HH and chorangiosis increase the risk of late fetal loss due to the high-output CHF.

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