Figures & data
Figure 1. Inhalational exposure to air pollutants like nitrous oxide (N2O) may spur alterations in opioidergic, glutamatergic, dopaminergic, and cholinergic activity, heightening parasympathetic dominance and reducing sympathetic activity, rendering susceptibility to cognitive deficits characteristic of ADHD and adverse metabolic effects, like obesity. α7nAChR: alpha 7 nicotinic acetylcholine receptor; DaR: dopamine receptor; NmdaR: NMDA receptor; Da: dopamine; DaT: dopamine transporter; KoR: kappa-opioid receptor; nNOS: neuronal nitric oxide synthase; NE: norepinephrine; N2O: nitrous oxide.
![Figure 1. Inhalational exposure to air pollutants like nitrous oxide (N2O) may spur alterations in opioidergic, glutamatergic, dopaminergic, and cholinergic activity, heightening parasympathetic dominance and reducing sympathetic activity, rendering susceptibility to cognitive deficits characteristic of ADHD and adverse metabolic effects, like obesity. α7nAChR: alpha 7 nicotinic acetylcholine receptor; DaR: dopamine receptor; NmdaR: NMDA receptor; Da: dopamine; DaT: dopamine transporter; KoR: kappa-opioid receptor; nNOS: neuronal nitric oxide synthase; NE: norepinephrine; N2O: nitrous oxide.](/cms/asset/895010c5-86fd-49fb-bea1-f1cc9bdf6811/ipgm_a_1189802_f0001_c.jpg)