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Clinical Features - Editorial

The possible role of air pollution in the link between ADHD and obesity

Pages 573-576 | Received 31 Jan 2016, Accepted 11 May 2016, Published online: 02 Jun 2016

Figures & data

Figure 1. Inhalational exposure to air pollutants like nitrous oxide (N2O) may spur alterations in opioidergic, glutamatergic, dopaminergic, and cholinergic activity, heightening parasympathetic dominance and reducing sympathetic activity, rendering susceptibility to cognitive deficits characteristic of ADHD and adverse metabolic effects, like obesity. α7nAChR: alpha 7 nicotinic acetylcholine receptor; DaR: dopamine receptor; NmdaR: NMDA receptor; Da: dopamine; DaT: dopamine transporter; KoR: kappa-opioid receptor; nNOS: neuronal nitric oxide synthase; NE: norepinephrine; N2O: nitrous oxide.

Figure 1. Inhalational exposure to air pollutants like nitrous oxide (N2O) may spur alterations in opioidergic, glutamatergic, dopaminergic, and cholinergic activity, heightening parasympathetic dominance and reducing sympathetic activity, rendering susceptibility to cognitive deficits characteristic of ADHD and adverse metabolic effects, like obesity. α7nAChR: alpha 7 nicotinic acetylcholine receptor; DaR: dopamine receptor; NmdaR: NMDA receptor; Da: dopamine; DaT: dopamine transporter; KoR: kappa-opioid receptor; nNOS: neuronal nitric oxide synthase; NE: norepinephrine; N2O: nitrous oxide.

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