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Review Article

Modulation of neuromuscular synapses and contraction in Drosophila 3rd instar larvae

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Pages 183-194 | Received 14 Mar 2018, Accepted 15 Jul 2018, Published online: 10 Oct 2018

Figures & data

Figure 1. Sites of modulation at arthropod neuromuscular synapses. Electrical impulses in the motor axons trigger release of the primary neurotransmitter, L-glutamate, from small synaptic vesicles. Effects of the primary transmitter are mediated by ionotropic receptors and can be modulated via GPCRs that are activated by co-transmitters and neurohormones.

Figure 1. Sites of modulation at arthropod neuromuscular synapses. Electrical impulses in the motor axons trigger release of the primary neurotransmitter, L-glutamate, from small synaptic vesicles. Effects of the primary transmitter are mediated by ionotropic receptors and can be modulated via GPCRs that are activated by co-transmitters and neurohormones.

Figure 2. Matrix-assisted laser desorption/ionization time of flight (MALDI-TOF) mass spectrometry of extracts from 10 Drosophila 3rd instar larvae reveals a small peak (arrow) at 649.6 Da, close to the predicted molecular mass of Proctolin (648 Da).

Figure 2. Matrix-assisted laser desorption/ionization time of flight (MALDI-TOF) mass spectrometry of extracts from 10 Drosophila 3rd instar larvae reveals a small peak (arrow) at 649.6 Da, close to the predicted molecular mass of Proctolin (648 Da).

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