Figures & data
Figure 1 The effect of acute exposure to ES and a SW on: (A) adherence, (B) zymosan particle phagocytosis and (C) PMA-primed H2O2 release from peritoneal macrophages isolated 24 h after stress. The values represent the mean (n = 8) ± SEM. Statistically significant differences: *, p < 0.05; and **, p < 0.01 vs. the IC group of rats.
Figure 2 The effect of in vitro treatment with: (A) CORT, 10− 9–10− 5 M; (B) NPY, 10− 14–10− 6 M; and (C) BE, 10− 14–10− 6 M on the adherence of macrophages isolated from IC rats, from rats exposed to acute ES and from rats exposed to a SW 24 h earlier. The values represent the mean (n = 8) ± SEM. Statistically significant differences: *, p < 0.05; **, p < 0.01; and ***, p < 0.001 vs. 0.
Figure 3 The effect of in vitro treatment with: (A) CORT, 10− 8–10− 5 M; (B) NPY, 10− 12–10− 6 M; and (C) BE, 10− 12–10− 6 M on zymosan phagocytosis by macrophages isolated from IC rats, from rats exposed to acute ES and from rats exposed to a SW 24 h earlier. The values represent the mean (n = 8) ± SEM. Statistically significant differences: *, p < 0.01; and **, p < 0.001 vs. 0.
Figure 4 The effect of in vitro treatment with: (A) CORT, 10− 9–10− 5 M; (B) NPY, 10− 14–10− 6 M; and (C) BE, 10− 14–10− 6 M on PMA-primed H2O2 release from macrophages isolated from IC rats, from rats exposed to acute ES and from rats exposed to a SW 24 h earlier. The values represent the mean (n = 6–8) ± SEM. Statistically significant differences: *, p < 0.05; **, p < 0.01; and ***, p < 0.0001 vs. 0.
