889
Views
4
CrossRef citations to date
0
Altmetric
Research Article

Effects of Chronic and Acute Pulmonary Hyperinflation on Phrenic Nerve Conduction in Patients with COPD

, , , , , & show all
Pages 378-383 | Received 15 Jan 2020, Accepted 02 Jun 2020, Published online: 25 Jun 2020

Figures & data

Table 1. Anthropometric characteristics and lung function parameters in healthy control subjects and all patients with COPD and in COPD patients divided according to the presence or absence of pulmonary hyperinflation at rest.

Figure 1. Individual values of mean latency (panel a) and mean amplitude (panel b) of cMAP in the stable COPD patients and matched control subjects enrolled in the study and subgroups of COPD patients divided according to the presence (IC < 80% pred.) or absence (IC ≥ 80% pred.) of pulmonary hyperinflation at rest. IC = Inspiratory Capacity.

Figure 1. Individual values of mean latency (panel a) and mean amplitude (panel b) of cMAP in the stable COPD patients and matched control subjects enrolled in the study and subgroups of COPD patients divided according to the presence (IC < 80% pred.) or absence (IC ≥ 80% pred.) of pulmonary hyperinflation at rest. IC = Inspiratory Capacity.

Figure 2. Correlation between values of mean latency of cMAP and resting IC (as % pred.) in stable COPD patients, suggesting an association between the prolonged velocity of conduction of phrenic nerves and degree of chronic pulmonary hyperinflation.

Figure 2. Correlation between values of mean latency of cMAP and resting IC (as % pred.) in stable COPD patients, suggesting an association between the prolonged velocity of conduction of phrenic nerves and degree of chronic pulmonary hyperinflation.

Table 2. Phrenic nerve conduction parameters in control subjects and all COPD patients, and in COPD patients divided according to the presence or absence of pulmonary hyperinflation at rest.

Figure 3. Individual values of amplitude and latency of cMAP during exacerbation (E) and at discharge (D) from the Hospital in a group of COPD patients hospitalized because of COPD exacerbation.

Figure 3. Individual values of amplitude and latency of cMAP during exacerbation (E) and at discharge (D) from the Hospital in a group of COPD patients hospitalized because of COPD exacerbation.

Table 3. Anthropometric characteristics, lung function parameters, and arterial blood gas values in a group of patients with COPD during exacerbation and at discharge.

Table 4. Phrenic nerve conduction parameters in a group of COPD patients during exacerbation and at discharge.

Reprints and Corporate Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

To request a reprint or corporate permissions for this article, please click on the relevant link below:

Academic Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

Obtain permissions instantly via Rightslink by clicking on the button below:

If you are unable to obtain permissions via Rightslink, please complete and submit this Permissions form. For more information, please visit our Permissions help page.