https://orcid.org/0000-0001-6722-8534
Figures & data
Figure 1. The common mechanisms of fungi that are involved in triggering pathogenesis, especially in oncogenesis. PRRs, pattern recognition receptors; CLRs, C-type lectin receptors; CARD9, Caspase Recruitment Domain-containing protein9; STAT1, signal transducer and activator of transcription 1; Th17, T helper 17 cell; IL-6, interleukin 6; IL-23, interleukin 23; TNF-α, tumor necrosis factor α; IL-12, interleukin 12; IFN-γ, interferon γ; IL-1β, interleukin 1β; MDSCs, myeloid-derived suppressor cells; TCL, cytotoxic T cell; ACH, acetaldehyde; MAPK, mitogen-activated protein kinases; EGFR, epithelial growth factor receptor; AhR, aryl hydrocarbon receptor; Hsp27, heat shock protein 27; G-CSF, granulocyte colony-stimulating factor; GM-CSF, granulocyte-macrophage colony-stimulating factor.
Figure 2. Fungi and their roles in specific tumors. Shown are the various mechanisms through which major gut fungi species are able to influence the specific cancers, including the immune responses, cytotoxins and metabolites, and bacterial–fungal interactions
Data availability statement
Data sharing is not applicable to this article as no new data were created or analyzed in this study.