Impact of anti‐oxidant status and apoptosis on the induction phase of chemotherapy in childhood acute lymphoblastic leukemia

Youssef Al‐TonbaryHematology/Oncology UnitMansoura University Children’sHospital, Mansoura, EgyptCorrespondence[email protected]

Samir Abou Al‐HasanHematology/Oncology UnitMansoura University Children’sHospital, Mansoura, Egypt

Maysaa ZakiHematology/Oncology UnitMansoura University Children’sHospital, Mansoura, Egypt

Ayman HammadHematology/Oncology UnitMansoura University Children’sHospital, Mansoura, Egypt

Shaimaa KandilHematology/Oncology UnitMansoura University Children’sHospital, Mansoura, Egypt

Ashraf FoudaHematology/Oncology UnitMansoura University Children’sHospital, Mansoura, Egypt

Figures & data

Figure 1. Different levels of MDA and TAC for patients at diagnosis and after 5 weeks of treatment in comparison to healthy controls: levels of apoptosis of patients at diagnosis and after 1 week of treatment are also shown.

Table 1. Clinical and laboratory data of patients at diagnosis

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Table 2. Comparison between patients who achieve remission and those who were resistant as regards MDA, TAC, and apoptosis at diagnosis and after therapy (Student’s t‐test)

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Table 3. Comparison between patients who lived and died (at the end of 5 weeks of treatment) as regards MDA, TAC, and apoptosis at diagnosis (Student’s t‐test)

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Impact of anti‐oxidant status and apoptosis on the induction phase of chemotherapy in childhood acute lymphoblastic leukemia

Table 1. Clinical and laboratory data of patients at diagnosis

Table 2. Comparison between patients who achieve remission and those who were resistant as regards MDA, TAC, and apoptosis at diagnosis and after therapy (Student’s t‐test)

Table 3. Comparison between patients who lived and died (at the end of 5 weeks of treatment) as regards MDA, TAC, and apoptosis at diagnosis (Student’s t‐test)

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Impact of anti‐oxidant status and apoptosis on the induction phase of chemotherapy in childhood acute lymphoblastic leukemia

Figures & data

Table 1. Clinical and laboratory data of patients at diagnosis

Table 2. Comparison between patients who achieve remission and those who were resistant as regards MDA, TAC, and apoptosis at diagnosis and after therapy (Student’s t‐test)

Table 3. Comparison between patients who lived and died (at the end of 5 weeks of treatment) as regards MDA, TAC, and apoptosis at diagnosis (Student’s t‐test)

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Academic Permissions

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