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Expert Review of Hematology Volume 2, 2009 - Issue 2
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Editorial

Hyperhemolysis syndrome in sickle cell disease

Nay Win Consultant Haematologist, National Blood Service Tooting Centre, 75 Cranmer Terrace, Tooting, London, SW17 0RB, UK. [email protected]
Pages 111-115 | Published online: 10 Jan 2014

Figures & data

Figure 1. Interaction and destruction of the patient’s own cells (HbSS/reticulocytes) and transfused cells by activated macrophages.

Hb: Hemoglobin; ICAM: Intercellular adhesion molecule; RBC: Red blood cell; VCAM: Vascular cell adhesion molecule.

Figure 1. Interaction and destruction of the patient’s own cells (HbSS/reticulocytes) and transfused cells by activated macrophages.Hb: Hemoglobin; ICAM: Intercellular adhesion molecule; RBC: Red blood cell; VCAM: Vascular cell adhesion molecule.

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Related Research Data

RBC ALLOIMMUNIZATION AND DEATH IN SCD
Source: Wiley
Successful management of the potentially fatal hyperhaemolysis syndrome of sickle cell anaemia with a regimen including bortezomib and Hemopure
Source: Wiley
Treatment of post‐transfusion hyperhaemolysis syndrome in Sickle Cell Disease with the anti‐IL6R humanised monoclonal antibody Tocilizumab
Source: Wiley
Posttransfusion hyperhemolysis is arrested by targeting macrophage activation with novel use of Tocilizumab.
Source: Wiley
A case-control analysis of hyperhemolysis syndrome in adults and laboratory correlates of complement involvement.
Source: Wiley
Letter to the Editor
Source: Wiley
Guidelines on red cell transfusion in sickle cell disease. Part I: principles and laboratory aspects
Source: Wiley
Editorial
Source: Wiley
Successful Outcome of Hyperhemolysis in Sickle Cell Disease following Multiple Lines of Treatment: The Role of Complement Inhibition
Source: Taylor & Francis

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