Advanced search
Publication Cover
Infection and Drug Resistance Volume 13, 2020 - Issue
Submit an article Journal homepage
150
Views
14
CrossRef citations to date
0
Altmetric
Review

Drugs for Multiple Sclerosis Activate Natural Killer Cells: Do They Protect Against COVID-19 Infection?

Mena Al-Ani1 Department of Clinical Sciences, College of Medicine and the Immuno-Oncology Group, Sharjah Institute for Medical Research, University of Sharjah, Sharjah27272, United Arab Emirates
,
Noha Mousaad Elemam1 Department of Clinical Sciences, College of Medicine and the Immuno-Oncology Group, Sharjah Institute for Medical Research, University of Sharjah, Sharjah27272, United Arab EmiratesORCID Iconhttps://orcid.org/0000-0002-3586-6731
ORCID Icon,
Jennifer Elisabeth Hundt2 Lübeck Institute for Experimental Dermatology, University of Lübeck, Lübeck, Germany
&
Azzam A Maghazachi1 Department of Clinical Sciences, College of Medicine and the Immuno-Oncology Group, Sharjah Institute for Medical Research, University of Sharjah, Sharjah27272, United Arab EmiratesCorrespondence[email protected]
ORCID Iconhttps://orcid.org/0000-0002-3310-678X
ORCID Icon
Pages 3243-3254 | Published online: 22 Sep 2020

Figures & data

Figure 1 Multiple sclerosis drugs as potential therapeutic agents for COVID-19. Drugs such as glatiramer acetate, vitamin D3, dimethyl fumarate, monomethyl fumarate, interferon-β, natalizumab or ocrelizumab, boost NK cell activity that could potentially eradicate SARS-CoV-2 virally infected cells.

Figure 1 Multiple sclerosis drugs as potential therapeutic agents for COVID-19. Drugs such as glatiramer acetate, vitamin D3, dimethyl fumarate, monomethyl fumarate, interferon-β, natalizumab or ocrelizumab, boost NK cell activity that could potentially eradicate SARS-CoV-2 virally infected cells.

Related research

People also read lists articles that other readers of this article have read.

Recommended articles lists articles that we recommend and is powered by our AI driven recommendation engine.

Cited by lists all citing articles based on Crossref citations.
Articles with the Crossref icon will open in a new tab.