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Review

Inflammatory mechanisms and oxidative stress in prostatitis: the possible role of antioxidant therapy

Pages 75-87 | Published online: 17 Sep 2018

Figures & data

Figure 1 Schematic illustration of the fundamentals involved in the mechanisms of acute prostatitis.

Abbreviations: IL, interleukin; iNOS, inducible nitric oxide synthase; MCP, monocyte chemoattractant protein; MIP, macrophage inflammatory protein; NF-kB, nuclear factor-kappa B; PRR, pattern-recognition receptor; RNS, reactive nitrogen species; ROS, reactive oxygen species; Th, T helper; TNF-α, tumor necrosis factor alpha.
Figure 1 Schematic illustration of the fundamentals involved in the mechanisms of acute prostatitis.

Figure 2 Schematic illustration of the fundamentals involved in the mechanisms of chronic prostatitis.

Abbreviations: EGF, epidermal growth factor; IFN, interferon; IL, interleukin; iNOS, inducible nitric oxide synthase; MCP, monocyte chemoattractant protein; NF-kB, nuclear factor-kappa B; RNS, reactive nitrogen species; ROS, reactive oxygen species; TGF, transforming growth factor; Th, T helper; TNF-α, tumor necrosis factor alpha; VEGF, vascular endothelial growth factor.
Figure 2 Schematic illustration of the fundamentals involved in the mechanisms of chronic prostatitis.