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Renal Failure Volume 38, 2016 - Issue 5
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Review

Endoplasmic reticulum stress and its effects on renal tubular cells apoptosis in ischemic acute kidney injury

Yan XuDepartment of Nephrology, Affiliated Hospital of Qingdao University, Qingdao, China;
,
Min GuoDepartment of Nephrology, Affiliated Hospital of Qingdao University, Qingdao, China;
,
Wei JiangDepartment of Nephrology, Affiliated Hospital of Qingdao University, Qingdao, China;
,
Hui DongDepartment of Nephrology, Affiliated Hospital of Qingdao University, Qingdao, China;
,
Yafei HanCenter of Medical Research, Affiliated Hospital of Qingdao University, Qingdao, China;
,
Xiao-Fei AnDepartment of Endocrinology, Jiangsu Province Hospital of Chinese Medicine, Nanjing, ChinaCorrespondence[email protected] [email protected]
&
Jisheng ZhangCenter of Medical Research, Affiliated Hospital of Qingdao University, Qingdao, China;
show all
Pages 831-837 | Received 08 Dec 2015, Accepted 26 Feb 2016, Published online: 22 Mar 2016

Figures & data

Figure 1. ER stress pathways. The accumulation of misfolded proteins in the ER activates the three typical signal pathways, PERK–eIF2–ATF4 pathway, IRE1–XBP1 pathway and ATF6 pathway as well as caspase 12.

Figure 1. ER stress pathways. The accumulation of misfolded proteins in the ER activates the three typical signal pathways, PERK–eIF2–ATF4 pathway, IRE1–XBP1 pathway and ATF6 pathway as well as caspase 12.

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