Figures & data
Figure 1. (a) HPTLC profile of CALE under UV 366 nm. (b) HPTLC chromatogram of CALE at a scan wavelength of 278 nm. T1: standard ellagic acid; T2: standard gallic acid; T3: standard quercetin.
![Figure 1. (a) HPTLC profile of CALE under UV 366 nm. (b) HPTLC chromatogram of CALE at a scan wavelength of 278 nm. T1: standard ellagic acid; T2: standard gallic acid; T3: standard quercetin.](/cms/asset/98af6226-b587-4170-9aa7-d4d197bd8f1e/iphb_a_876052_f0001_b.jpg)
Figure 2. Effect of CALE on H+K+ATPase activity in gastric mucosa and gastric wall mucus in EtOH-induced ulcer group. +p < 0.001 compared to the respective normal control group. ap < 0.05, bp < 0.01 and cp < 0.001 compared to the respective EtOH-induced ulcer group.
![Figure 2. Effect of CALE on H+K+ATPase activity in gastric mucosa and gastric wall mucus in EtOH-induced ulcer group. +p < 0.001 compared to the respective normal control group. ap < 0.05, bp < 0.01 and cp < 0.001 compared to the respective EtOH-induced ulcer group.](/cms/asset/052d5e63-959b-44e9-9fdc-b6b7aa01dc79/iphb_a_876052_f0002_b.jpg)
Table 1. Gastroprotective activity of CALE on aspirin-, EtOH-, CRS-, and PL-induced ulcers.
Table 2. Effect of CALE on gastric secretion in 4h PL rats: effect on volume, acid, and pepsin.