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Meeting Report

A fruitful chromatin harvest: Meeting summary of the Second European Workshop on Plant Chromatin 2011 in Versailles, France

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Pages 307-311 | Published online: 01 Mar 2012

Figures & data

Figure 1. Localization of the 5S (red) and 45S (green) loci in interphase nuclei of wild-type (A) and fas (B) mutant of A. thaliana by Fluorescent in situ hybridization (FISH) showing a 45S rDNA loss in the fas mutant. Courtesy of J. Fajkus (Brno, Czech Republic).

Figure 1. Localization of the 5S (red) and 45S (green) loci in interphase nuclei of wild-type (A) and fas (B) mutant of A. thaliana by Fluorescent in situ hybridization (FISH) showing a 45S rDNA loss in the fas mutant. Courtesy of J. Fajkus (Brno, Czech Republic).

Figure 2. Model of AtBMI1C regulation during plant development. AtBMI1C is an endosperm imprinted gene. The expression of the maternal allele depends on DNA demethylation and a putative transcriptional activator/s, and the silencing of the paternal allele requires high levels of maternal transcripts, CG methylation, and RdDM. AtBMI1C is not expressed during embryo or vegetative development. The silencing in young embryos is achieved through an as-yet unknown mechanism, but in mature embryos and then after germination, the repression of AtBMI1C depends on PcG. During stamen development, the PcG-mediated repression of AtBMI1C is released to allow the biallelic expression of the gene. Courtesy of M. Calonje (Heidelberg, Germany).

Figure 2. Model of AtBMI1C regulation during plant development. AtBMI1C is an endosperm imprinted gene. The expression of the maternal allele depends on DNA demethylation and a putative transcriptional activator/s, and the silencing of the paternal allele requires high levels of maternal transcripts, CG methylation, and RdDM. AtBMI1C is not expressed during embryo or vegetative development. The silencing in young embryos is achieved through an as-yet unknown mechanism, but in mature embryos and then after germination, the repression of AtBMI1C depends on PcG. During stamen development, the PcG-mediated repression of AtBMI1C is released to allow the biallelic expression of the gene. Courtesy of M. Calonje (Heidelberg, Germany).

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