Figures & data
Figure 1 Model of the Fe deficiency effects on the metabolism of cucumber root cells. In the control condition (A) mitochondria are able to satisfy the energetic and the NAD(P)H turnover requests of the cell. In Fe deficiency (-Fe) condition (B), the ATP and NAD(P)H request are enhanced and the respiratory chain is strongly impaired leading the cellular metabolism to change several pathways. To overcome the energetic emergency status, the cell increases (i) glycolysis; (ii) the synthesis of DTC protein leading to an enhanced of citrate/malate exchange between mitochondrial matrix and cytosol; (iii) the anaerobic metabolism. Changes in the arrow thickness indicates the changes in the rate of metabolic processes occurring under Fe deficiency condition. Abbreviations: FC-R, Ferric chelate-reductase; IRT, iron regulated transporter; DTC, di-tricarboxylic acid carrier; PM, plasma membrane.
![Figure 1 Model of the Fe deficiency effects on the metabolism of cucumber root cells. In the control condition (A) mitochondria are able to satisfy the energetic and the NAD(P)H turnover requests of the cell. In Fe deficiency (-Fe) condition (B), the ATP and NAD(P)H request are enhanced and the respiratory chain is strongly impaired leading the cellular metabolism to change several pathways. To overcome the energetic emergency status, the cell increases (i) glycolysis; (ii) the synthesis of DTC protein leading to an enhanced of citrate/malate exchange between mitochondrial matrix and cytosol; (iii) the anaerobic metabolism. Changes in the arrow thickness indicates the changes in the rate of metabolic processes occurring under Fe deficiency condition. Abbreviations: FC-R, Ferric chelate-reductase; IRT, iron regulated transporter; DTC, di-tricarboxylic acid carrier; PM, plasma membrane.](/cms/asset/d3969c1f-3a51-4b8d-ae9d-6efc4c978364/kpsb_a_10908344_f0001.gif)