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Correction

Correction

This article refers to:
Contribution of nucleophosmin overexpression to multidrug resistance in breast carcinoma

Article title: “Contribution of nucleophosmin overexpression to multidrug resistance in breast carcinoma”

Authors: Siying Chen, Ti Meng, Xiaowei Zheng, Jiangxia Cai, Weipeng Zhang, Haisheng You, Jianfeng Xing and Yalin Dong

Journal: Journal of Drug Targeting

Citation details: Volume 26, Number 1, pages 27–35

DOI: https://doi.org/10.1080/1061186X.2017.1332066

When the above article was published online and in print, the is incorrect. In addition, a reference to our previous research is missing.

Below is the corrected version of and the citation to our previously published article has been inserted in the references.

Figure 2. Methotrexate-mediated resistance promotes the activation of PI3K/Akt pathway and impede cell apoptosis. Western blot detection (A) and quantitative analysis (B) of NPM, PTEN, p-Akt, t-Akt, Bcl-2 and Bax both in MCF-7/MTX cells under methotrexate treatment (2.8 μM). Relative protein expressions were presented as mean ± SD, β-actin was used as loading control, *P < 0.05, **P < 0.01 by ANOVA versus control group.

Figure 2. Methotrexate-mediated resistance promotes the activation of PI3K/Akt pathway and impede cell apoptosis. Western blot detection (A) and quantitative analysis (B) of NPM, PTEN, p-Akt, t-Akt, Bcl-2 and Bax both in MCF-7/MTX cells under methotrexate treatment (2.8 μM). Relative protein expressions were presented as mean ± SD, β-actin was used as loading control, *P < 0.05, **P < 0.01 by ANOVA versus control group.

This article is available both online and in print. The online version has been corrected.

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