2,975
Views
20
CrossRef citations to date
0
Altmetric
Research Article

RETRACTED ARTICLE: Mechanism of MALAT1 preventing apoptosis of vascular endothelial cells induced by oxygen–glucose deficiency and reoxidation

, &
Pages 798-805 | Received 06 Dec 2017, Accepted 30 Jan 2018, Published online: 26 Mar 2018
 

Abstract

We, the Editors and Publisher of the journal Artificial Cells, Nanomedicine, and Biotechnology, have retracted the following article: 

Guoping Wang, Yuanbo Wu & Yuyou Zhu (2018) Mechanism of MALAT1 preventing apoptosis of vascular endothelial cells induced by oxygen–glucose deficiency and reoxidation. Artificial Cells, Nanomedicine, and Biotechnology, 46(sup1), 798–805, DOI: 10.1080/21691401.2018.1436065

Since publication, the authors repeated some of their experiments and found that under the same conditions they could not repeat the results reported in this paper. In addition, concerns were raised regarding the high degrees of similarity with the following previously published paper: Jia-Wei Xin & Yu-gang Jiang (2017) Long noncoding RNA MALAT1 inhibits apoptosis induced by oxygen-glucose deprivation and reoxygenation in human brain microvascular endothelial cells, Experimental and Therapeutic Medicine, 13(4), 1225–1234, DOI: 10.3892/etm.2017.4095. As this directly impacts the integrity of the reported results and conclusions, the authors alerted the issue to the Editor and Publisher and all have agreed to retract the article to ensure correction of the scholarly record.

We have been informed in our decision-making by our policy on publishing ethics and integrity and the COPE guidelines on retractions.

The retracted article will remain online to maintain the scholarly record, but it will be digitally watermarked on each page as “Retracted”.

View retraction statement:
Statement of Retraction

Reprints and Corporate Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

To request a reprint or corporate permissions for this article, please click on the relevant link below:

Academic Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

Obtain permissions instantly via Rightslink by clicking on the button below:

If you are unable to obtain permissions via Rightslink, please complete and submit this Permissions form. For more information, please visit our Permissions help page.