A 56-year-old fireman previously revascularized by coronary artery bypass presented unable to work 9 years following index surgery. Investigations revealed extrinsic compression of the right ventricular outflow tract between a coronary vein graft aneurysm and the ascending aorta (). Invasive systolic right ventricular pressure was elevated (83 mmHg peak pressure), with dynamic outflow tract obstruction (, Supplemental Video 1). Following heart team discussion in the absence of distal run off from the graft aneurysm the patient was offered transcatheter closure of the feeding vessel. The vein graft was cannulated with a JR4 catheter, sized (), and an 0.035ʹʹ Terumo wire used to advance distally to position an Amplatzer superstiff wire in the aneurysm. A 5F Amplatzer Torquevue 2 delivery system was used to deploy an 8 mm AVPII device within the proximal part of the vein graft. Non-invasive follow-up over the next 12 months showed resolution of acquired RVOT obstruction. The patient returned to work at 6 months
Stenosis of a tube is caused by internal, intramural, or external factors. Compression of the main pulmonary artery has previously been reported due to giant aneurysms of the native coronary vessels, pathologies of the aortic root (unruptured sinus of val salva aneurysm,Citation1 and dissection), and mediastinal masses. Isolated case reports of acquired obstruction post-congenitalCitation2 or adultCitation3 cardiac surgery involving the great vessels are also known but this case adds to the list of cardiac structures that may be compressed by coronary graft aneurysm, percutaneous treatment of which appears to be uncomplicated and effective.
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