The Effects of D-Tubocurarine, Hexamethonium and Decamethonium on the Receptor for the Axon Reflex Sweating

Masao Wada Physiological Laboratory of Prof. M. Wada, Toholcu University School of Medicine, Sendai

Toshio Nakagawa Physiological Laboratory of Prof. M. Wada, Toholcu University School of Medicine, Sendai

Norio Hanawoka Physiological Laboratory of Prof. M. Wada, Toholcu University School of Medicine, Sendai

Koshiro Hatanaka Physiological Laboratory of Prof. M. Wada, Toholcu University School of Medicine, Sendai

Hiroshi Funato Physiological Laboratory of Prof. M. Wada, Toholcu University School of Medicine, Sendai

Saburo Kanazawa Physiological Laboratory of Prof. M. Wada, Toholcu University School of Medicine, Sendai

Nobuyoshi Morikawa Physiological Laboratory of Prof. M. Wada, Toholcu University School of Medicine, Sendai

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Abstract

Beginning with the work of Coon and Rothman in 1939–41, it has been substantiated that nicotine and nicotine-like agents produce the axon reflex sweating in human skin. The sweating of the same mechanism can be elicited also by high concentrations of sodium chloride (Wada, Arai, Takagaki and Nakagawa, 1952).

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The Effects of D-Tubocurarine, Hexamethonium and Decamethonium on the Receptor for the Axon Reflex Sweating

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The Effects of D-Tubocurarine, Hexamethonium and Decamethonium on the Receptor for the Axon Reflex Sweating

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