Interaction of Psychoactive Drugs with Exposure to Electromagnetic Fields

Abstract

Evidence has previously been presented to show that the dopamine (DA) and opiate systems of the brain, in part, mediate the effects of exposure to electromagnetic (EM) fields. The hypothesis was broadened to predict that low-intensity EM energy exposure would interact with naloxone, chlor-diazepoxide and haloperidol treatment in tests using analgesia and stereotypic behavior. In the set of experiments reported here, the data obtained indicated there were such interactions.

One implication of the dopamine-opiate hypothesis is that EM energy exposure would likely affect the hypothalamic set-point for body temperature regulation. Substantial evidence for this view now exits. This theory and data indicate that body temperature rise with exposure is likely to be a consequence of the energy affecting the dopamine-opiate systems of the brain.