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Erratum

Erratum to: Alers S, Wesselborg S, Stork B. ATG13: Just a companion, or an executor of the autophagic program? Autophagy 2014; 10:944–56

, &
Page 1481 | Received 20 Jun 2014, Accepted 20 Jun 2014, Published online: 24 Jun 2014
This article refers to:
ATG13

In our review article “ATG13: Just a companion, or an executor of the autophagic program?” (Alers S, et al., Autophagy 10:6, 944–56), we stated: “Notably, no Drosophila or C. elegans homolog of Atg17 or RB1CC1 has been described so far.” This statement is incorrect. Two papers described the identification and characterization of Drosophila Atg17/RB1CC1/FIP200 and established that this fly gene is the homolog of the corresponding human gene (Kim M, et al., Autophagy 9:8, 1201–13Citation1 and Nagy P, et al., Autophagy 10:3, 453–67Citation2). We would like to apologize to the readership of Autophagy in general and to the authors of those 2 manuscripts in particular for this erroneous statement.

Disclosure of Potential Conflicts of Interest

No potential conflicts of interest were disclosed.

References

  • Kim M, Park HL, Park HW, Ro SH, Nam SG, Reed JM, Guan JL, Lee JH. Drosophila Fip200 is an essential regulator of autophagy that attenuates both growth and aging. Autophagy 2013; 9:1201 - 13; http://dx.doi.org/10.4161/auto.24811; PMID: 23819996
  • Nagy P, Kárpáti M, Varga A, Pircs K, Venkei Z, Takáts S, Varga K, Érdi B, Hegedűs K, Juhász G. Atg17/FIP200 localizes to perilysosomal Ref(2)P aggregates and promotes autophagy by activation of Atg1 in Drosophila. Autophagy 2014; 10:453 - 67; http://dx.doi.org/10.4161/auto.27442; PMID: 24419107