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Research Article

Cochlear Distribution of Na,K-ATPase and Corticosteroid Receptors in Two Mouse Strains with Congenital Hearing Disorders

Pages 794-802 | Published online: 08 Jul 2009

  • Yao X, ten Gate WJF, Curtis LM, Rarey KE. Expression of Na + , K + -ATPase al subunit mRNA in the developing rat cochlea. Hear Res 1994: 80: 31-7.
  • Bosher SK. The effects of inhibition of the striai NaK-activated ATPase by perilymphatic ouabain in the guinea pig. Acta Otolaryngol (Stockh) 1980: 90: 21929.
  • Rarey KE, Luttge WG. Presence of type I and type II/IB receptors for adrenocorticosteroid hormones in the inner ear. Hear Res 1989: 41: 217-22.
  • Rarey KE, Tyneway D, Patterson K. Decreased adenosine triphosphatase activity in the absence of adrenocorticosteroids. Arch Otolaryngol Head Neck Surg 1989; 115: 817-21.
  • Curtis LM, ten Cate WJF, Rarey KE. Dynamics of Na.K-ATPase sites in the lateral cochlear wall tissues of the rat. Eur Arch Otorhinolaryngol 1993; 250: 265-70.
  • Probst FJ, Camper SA. The role of mouse mutants in the identification of human hereditary hearing loss genes. Hear Res 1999; 130: 1-6.
  • Friedmann TB, Liang Y, Weber JL, et al. A gene for congenital, recessive deafness DFNB3 maps to the pericentromeric region of chromosome 17. Nat Genet 1995: 9: 86-91.
  • Wang A, Liang Y, Fridell RA, et al. Association of unconventional myosin MYO15 mutations with human nonsyndromic deafness DFNB3. Science 1998: 280: 1447-51.
  • Anniko M. Specific pathology of the stria vascularis in postnatal progressive genetic inner ear disorder. Hear Res 1982; 6: 247-58.
  • Anniko M, Wroblewski R. Deterioration of the elemental composition of endolymph in genetic inner ear disease. Arch Otorhinolaryngol 1980: 228: 171-86.
  • Hultcrantz M, Anniko M. Influence of gamma irradiation on developing otoconia. Am J Otolaryngol 1985: 6: 79-91.
  • Spicer SS, Schulte BA. Evidence for a medial K + recycling pathway from inner hair cells. Hear Res 1998: 118: 1-12.
  • Kuijpers W. Na-K-ATPase activity in the cochlea of the rat during development. Acta Otolaryngol (Stockh) 1974: 78: 341-4.
  • Erichsen S, Zuo J, Curtis L, Rarey K, Hultcrantz M. Na.K-ATPase a- and ß-isoforms in the developing inner ear of the mouse. Hear Res 1996; 100: 143-9.
  • Gratton MA, Smyth BJ, Lam CF, Boettcher FA, Schmied RA. Decline in the endocochlear potential corresponds to decreased Na.K-ATPase activity in the lateral wall of quiet-aged gerbils. Hear Res 1997: 108: 9-16.
  • Probst FJ, Fridell RA, Raphael Y, et al. Correction of deafness in shaker-2 mice by an unconventional myosin in a BAC transgene. Science 1998; 280: 14447.
  • Liang Y, Wang A, Belyantseva IA, et al. Characterization of the human and mouse unconventional myosin XV genes responsible for hereditary deafness DFNB3 and Shaker 2. Genomics 1999: 61: 243-58.
  • Lohuis PJMF, ten Cate WJF, Patterson KE, Rarey KE. Modulation of the rat stria vascularis in the absence of circulating adrenocorticosteroids, Acta Otolaryngol (Stockh) 1990: 110: 348-56.
  • Pitovski DZ, Drescher MJ, Kerr TP, Drescher DG. Aldosterone mediates an increase in [3H]ouabai n binding at Na+ ,K +-ATPase sites in the mammalian inner ear. Hear Res 1993: 601: 273-8.
  • Erichsen S, Bagger-Sjöbäck D, Curtis L, Zuo J, Rarey K, Hultcrantz M. Appearance of glucocorticoid receptors in the inner ear of the mouse during development. Acta Otolaryngol (Stockh) 1996: 116: 721-5.
  • Erichsen S, Stierna P, Bagger-Sjöbäck D, et al. Distribution of Na.K-ATPase is normal in the inner ear of a mouse with a null mutation of the glucocorticoid receptor. Hear Res 1998; 124: 146-54.
  • Ferrary E, Bernard C, Teixeira M, et al. Hormonal modulation of inner ear fluids. Acta Otolaryngol (Stockh) 1996: 116: 244-7.
  • Berger S, Bleich M, Schmid W, et al. Mineralocorticoid receptor knockout mice: pathophysiology of Na+ metabolism. Proc Natl Acad Sei USA 1998; 95: 9424-9.

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