https://orcid.org/0000-0002-6884-968X
References
- Ghosh S. Cisplatin: the first metal based anticancer drug. Bioorg Chem. 2019;88:102925.
- Waissbluth S, Daniel SJ. Cisplatin-induced ototoxicity: transporters playing a role in cisplatin toxicity. Hear Res. 2013;299:37–45.
- Jamieson ER, Lippard SJ. Structure, recognition, and processing of cisplatin-DNA adducts. Chem Rev. 1999;99(9):2467–2498.
- Rybak LP, Mukherjea D, Ramkumar V. Mechanisms of cisplatin-induced ototoxicity and prevention. Semin Hear. 2019;40(2):197–204.
- So H, Kim H, Lee JH, et al. Cisplatin cytotoxicity of auditory cells requires secretions of proinflammatory cytokines via activation of ERK and NF-kappaB. J Assoc Res Otolaryngol. 2007;8(3):338–355.
- van Ruijven MW, de Groot JC, Hendriksen F, et al. Immunohistochemical detection of platinated DNA in the cochlea of cisplatin-treated Guinea pigs. Hear Res. 2005;203(1–2):112–121.
- Ciarimboli G, Deuster D, Knief A, et al. Organic cation transporter 2 mediates cisplatin-induced oto- and nephrotoxicity and is a target for protective interventions. Am J Pathol. 2010;176(3):1169–1180.
- Manohar S, Jamesdaniel S, Ding D, et al. Quantitative PCR analysis and protein distribution of drug transporter genes in the rat cochlea. Hear Res. 2016;332:46–54.
- More SS, Akil O, Ianculescu AG, et al. Role of the copper transporter, CTR1, in platinum-induced ototoxicity. J Neurosci. 2010;30(28):9500–9509.
- Hellberg V, Gahm C, Liu W, et al. Immunohistochemical localization of OCT2 in the cochlea of various species. Laryngoscope. 2015;125(9):E320–5.
- Fransson AE, Kisiel M, Pirttilä K, et al. Hydrogen inhalation protects against ototoxicity induced by intravenous cisplatin in the Guinea pig. Front Cell Neurosci. 2017;11:280.
- Thomas AJ, Hailey DW, Stawicki TM, et al. Functional mechanotransduction is required for cisplatin-induced hair cell death in the zebrafish lateral line. J Neurosci. 2013;33(10):4405–4414.
- Nakamura T, Yonezawa A, Hashimoto S, et al. Disruption of multidrug and toxin extrusion MATE1 potentiates cisplatin-induced nephrotoxicity. Biochem Pharmacol. 2010;80(11):1762–1767.
- Staud F, Cerveny L, Ahmadimoghaddam D, et al. Multidrug and toxin extrusion proteins (MATE/SLC47); role in pharmacokinetics. Int J Biochem Cell Biol. 2013;45(9):2007–2011.
- Estrela GR, Wasinski F, Felizardo RJF, et al. MATE-1 modulation by kinin B1 receptor enhances cisplatin efflux from renal cells. Mol Cell Biochem. 2017;428(1–2):101–108.
- Freitas-Lima LC, Budu A, Arruda AC, et al. PPAR-α deletion attenuates cisplatin nephrotoxicity by modulating renal organic transporters MATE-1 and OCT-2. Int J Mol Sci. 2020;21(19):7416.
- Teft WA, Winquist E, Nichols AC, et al. Predictors of cisplatin-induced ototoxicity and survival in chemoradiation treated head and neck cancer patients. Oral Oncol. 2019;89:72–78.
- Abitbol J, Beach R, Barr K, et al. Cisplatin-induced ototoxicity in organotypic cochlear cultures occurs independent of gap junctional intercellular communication. Cell Death Dis. 2020;11(5):342.
- Jensen R, Glazer PM. Cell-interdependent cisplatin killing by Ku/DNA-dependent protein kinase signaling transduced through gap junctions. Proc Natl Acad Sci U S A. 2004;101(16):6134–6139.
- Li J, Liu C, Kaefer S, et al. The mechanotransduction channel and organic cation transporter are critical for cisplatin ototoxicity in murine hair cells. Front Mol Neurosci. 2022;15:835448.