Hypercoagulability

References

• Heijboer H, Brandjes DP, Büller HR, et al. Deficiencies of coagulation-inhibiting and fibrinolytic proteins in outpatients with deep-vein thrombosis. N Engl J Med 1990: 323(22): 1512–6
   PubMed Web of Science ®Google Scholar
• Moore TJ. Effect of partial ileal bypass surgery on coronary heart disease. (Letter) N Engl J Med 1991: 324(8): 563
   Google Scholar
• Thaler E, Lechner K. Antithrombin III deficiency and thromboembolism. Clin Haematol 1981: 10(2): 369–90
   PubMedGoogle Scholar
• Hirsh J. Laboratory diagnosis of thrombosis. In: Colman RW, Hirsh J, Marder VJ, et al, eds. Hemostasis and thrombosis: basic principles and clinical practice. 2d ed. Philadelphia: JB Lippincott, 1987: 1165–83
   Google Scholar
• Rosenberg RD. Biologic actions of hepar in. Semin Hematol 1977: 14(4): 427–40
   PubMed Web of Science ®Google Scholar
• Rosenberg RD, Dam us PS. The purification and mechanism of action of human antithrombin-heparin cofactor. J Biol Chem 1973: 248(18): 6490–505
   PubMed Web of Science ®Google Scholar
• Tollefsen DM. Laboratory diagnosis of anti-thrombin and heparin cofactor II deficiency. Semin Thromb Hemost 1990: 16(2): 162–8
   PubMed Web of Science ®Google Scholar
• Miletich JP. Laboratory diagnosis of protein C deficiency. Semin Thromb Hemost 1990: 16(2): 169–76
   PubMed Web of Science ®Google Scholar
• Comp PC, Nixon RR, Cooper MR, et al. Familial protein S deficiency is associated with recurrent thrombosis. J Clin Invest 1984: 74(6): 2082–8
   PubMed Web of Science ®Google Scholar
• Comp PC, Doray D, Palton D, et al. An abnormal plasma distribution of protein S occurs in functional protein S deficiency. Blood 1986: 67(2): 504–8
   PubMed Web of Science ®Google Scholar
• Engesser L, Broekmans AW, Briet E, et al. Hereditary protein S deficiency: clinical manifestations. Ann Intern Med 1987: 106(5): 677–82
   PubMed Web of Science ®Google Scholar
• Broekmans AW, Veitkamp JJ, Bertina RM. Congenital protein C deficiency and venous thromboembolism: a study of three Dutch families. N EnglJ Med 1983: 309(6): 340–4
   PubMed Web of Science ®Google Scholar
• Schrader J. Antithrombin III in renal disease: importance and treatment. In: Bottinger LE, ed. Antithrombin deficiency. Stockholm: KabiVitrum, 1988: 43–7
   Google Scholar
• Bottinger LE, ed. Antithrombin deficiency. Stockholm: KabiVitrum, 1988
   Google Scholar
• Miletich J, Sherman L, Broze G Jr. Absence of thrombosis in subjects with heterozygous protein C deficiency. N Engl J Med 1987: 317(16): 991–6
   PubMed Web of Science ®Google Scholar
• Comp PC. Laboratory evaluation of protein S status. Semin Thromb Hemost 1990: 16(2): 177–81
   PubMed Web of Science ®Google Scholar
• D'Angelo A, Vigano-D'Angelo S, Esmon CT, et al. Acquired deficiencies of protein S: protein S activity during oral anticoagulation, in liver disease, and in disseminated intravascular coagulation. J Clin Invest 1988: 81(5): 1445–54
   PubMed Web of Science ®Google Scholar
• Griffin JH, Evatt B, Zimmerman TS, et al. Deficiency of protein C in congenital thrombotic disease. J Clin Invest 1981: 68(5): 1370–3
   PubMed Web of Science ®Google Scholar
• Nilsson IM, Ljungner H, Tengborn L. Two different mechanisms in patients with venous thrombosis and defective fibrinolysis: low concentration of plasminogen activator or increased concentration of plasminogen activator inhibitor. BMJ (Clin Res) 1985: 290(6480): 1453–6
   PubMedGoogle Scholar
• Kruithof EK. Plasminogen activator inhibitor type I: biochemical, biological, and clinical aspects. Fibrinolysis 1988: 2(Suppl 2): 59–70
   Google Scholar
• Bachmann F. Laboratory diagnosis of impairment of fibrinolysis in patients with thromboembolic disease. Semin Thromb Hemost 1990: 16(2): 193–7
   PubMed Web of Science ®Google Scholar
• D'Angelo A, Kluft C, Verheijen JH, et al. Fibrinolytic shut-down after surgery: impairment of the balance between tissue-type plasminogen activator and its specific inhibitor. Eur J Clin Invest 1985: 15(6): 308–12
   PubMed Web of Science ®Google Scholar
• Rocha E, Alfaro MJ, Paramo JA, et al. Preoperative identification of patients at high risk of deep venous thrombosis despite prophylaxis in total hip replacement. Thromb Haemost 1988: 59(1): 93–5
   PubMed Web of Science ®Google Scholar
• Hamsten A, de Faire U, Walldius G, et al. Plasminogen activator inhibitor in plasma: risk factor for recurrent myocardial infarction. Lancet 1987: 2(8549): 3–9
   PubMed Web of Science ®Google Scholar
• Korninger C, Lechner K, Niessner H, et al. Impaired fibrinolytic capacity predisposes for recurrence of venous thrombosis. Thromb Haemost 1984: 52(2): 127–30
   PubMed Web of Science ®Google Scholar
• Triplett DA. Laboratory diagnosis of lupus anticoagulants. Semin Thromb Hemost 1990: 16(2): 182–92
   PubMed Web of Science ®Google Scholar
• Bowie EJ, Thompson JH Jr, Pascuzzi CA, et al. Thrombosis in systemic lupus erythematosus despite circulating anticoagulants. J Lab Clin Med 1963: 62(Sep): 416–30
   PubMedGoogle Scholar
• Lechner K. Lupus anticoagulants and thrombosis. In: Verstaete M, Lijnen R, Arnout J, eds. Thrombosis and haemostasis. Leuven, Belgium: Leuven Univ Press, 1987: 525–47
   Google Scholar
• Barbui T, Cortelazzo S, Galli M, et al. Lupus anticoagulant and repeated abortions: a case control study. (Abstr) Thromb Haemost 1987: 58(1): 232
   Web of Science ®Google Scholar
• Howard MA, Firkin BG, Healy DL, et al. Lupus anticoagulant in women with multiple spontaneous miscarriage. Am J Hematol 1987: 26(2): 175–8
   PubMed Web of Science ®Google Scholar
• Brandt JT, Triplett DA. The effect of phospholipid on the detection of lupus anticoagulants by the dilute Russell viper venom time. Arch Pathol Lab Med 1989: 113(12): 1376–8
   PubMed Web of Science ®Google Scholar
• Brandt JT, Triplett DA, Rock WA, et al. Effect of lupus anticoagulants on the activated partial thromboplastin time. Results of College of American Pathologists survey program. Arch Pathol Lab Med 1991: 115(2): 109–14
   PubMed Web of Science ®Google Scholar