References
- He SH. Key role of mast cells and their major secretory products in inflammatory bowel disease. World J Gastroenterol 2004; 10: 309–18
- Raithel M, Matek M, Baenkler HW, Jorde W, Hahn EG. Mucosal histamine content and histamine secretion in Crohn's disease, ulcerative colitis and allergic enteropathy. Int Arch Allergy Immunol 1995; 108: 127–33
- Weidenhiller M, Raithel M, Winterkamp S, Otte P, Stolper J, Hahn EG. Methylhistamine in Crohn's disease (CD): increased production and elevated urine excretion correlates with disease activity. Inflamm Res 2000; 49 Suppl 1: S35–6
- Winterkamp S, Weidenhiller M, Otte P, Stolper J, Schwab D, Hahn EG, et al. Urinary excretion of N-methylhistamine as a marker of disease activity in inflammatory bowel disease. Am J Gastroenterol 2002; 97: 3071–7
- Bischoff SC, Grabowsky J, Manns MP. Quantification of inflammatory mediators in stool samples of patients with inflammatory bowel disorders and controls. Dig Dis Sci 1997; 42: 394–403
- Fox CC, Lichtenstein LM, Roche JK. Intestinal mast cell responses in idiopathic inflammatory bowel disease. Histamine release from human intestinal mast cells in response to gut epithelial proteins. Dig Dis Sci 1993; 38: 1105–12
- Maintz L, Novak N. Histamine and histamine intolerance. AJCN 2007; 85: 1185–96
- Barbry P, Champe M, Chassande O, Munemitsu S, Champigny G, Lingueglia E, et al. Human kidney amiloride-binding protein: cDNA structure and functional expression. Proc Natl Acad Sci USA 1990; 87: 7347–518
- García-Martín E, Ayuso P, Martínez C, Blanca M, Agúndez JA. Histamine pharmacogenomics. Pharmacogenomics 2009; 10: 867–83
- NCBI. Available at, , http://www.ncbi.nlm.nih.gov/SNP/snp_ref.cgi?rs=rs1049793.
- Ayuso P, Garcia-Martin E, Martinez C, Agundez JA. Genetic variability of human diamine oxidase: occurrence of three nonsynonymous polymorphisms and study of their effect on serum enzyme activity. Pharmacogenet Genomics 2007; 17: 687–93
- Garcia-Martin E, Mendoza JL, Martinez C, Taxonera C, Urcelay E, Ladero JM. Severity of ulcerative colitis is associated with a polymorphism at diamine oxidase gene but not at histamine N-methyltransferase gene. World J Gastroenterol 2006; 12: 615–20
- Lennard-Jones JE. Classification of inflammatory bowel disease. Scand J Gastroenterol 1989; 24 Suppl 170: 2–6
- Silverberg MS, Satsagi J, Ahmad T, Arnott ID, Nernstein CN, Brant SR, et al. Toward an integrated clinical, molecular and serological classification of inflammatory bowel disease: Report of a Working Party of the 2005 Montreal World Congress of Gastroenterology. Can J Gastroenterol 2005; 19 Suppl A: 5–36
- Dvorak AM, Monahan RA, Osage JE, Dickersin GR. Crohn's disease: transmission electron microscopic studies. II. Immunologic inflammatory response. Alterations of mast cells, basophils, eosinophils, and the microvasculature. Hum Pathol 1980; 11: 606–19
- Gelbmann CM, Mestermann S, Gross V, Köllinger M, Schölmerich J, Falk W. Strictures in Crohn's disease are characterised by an accumulation of mast cells colocalised with laminin but not with fibronectin or vitronectin. Gut 1999; 45: 210–7
- Knutson L, Ahrenstedt O, Odlind B, Hällgren R. The jejunal secretion of histamine is increased in active Crohn's disease. Gastroenterology 1990; 98: 849–54
- García-Martín E, Ayuso P, Martínez C, Agúndez JA. Improved analytical sensitivity reveals the occurrence of gender-related variability in diamine oxidase enzyme activity in healthy individuals. Clin Biochem. 2007; 40: 1339–41