Effect of acute acid loading on acid–base and calcium metabolism

References

• Andersen DA. Environmental factors in the aetiology of urolithiasis. Urinary calculi, LC Delatte, A Rapado, A Hodgkinson. Karger, BasleSwitzerland 1973; 7–21
   Google Scholar
• Robertson WG, Peacock M, Heyburn PJ, Hanes FA. Epidemiological risk factors in calcium stone disease. Scand J Urol Nephrol Suppl 1980; 53: 15–28
   PubMedGoogle Scholar
• Iguchi M, Umekawa T, Ishikawa Y, Katayama Y, Kodama M, Tkada M, et al. Dietary intake and habits of Japanese renal stone patients. J Urol 1990; 143: 1093–5
   PubMed Web of Science ®Google Scholar
• Robertson WG, Heyburn PJ, Peacock M, Hanes FA, Swaminathan R. The effect of high animal protein intake on the risk of calcium stone formation in the urinary tract. Clin Sci 1979; 57: 285–8
   PubMed Web of Science ®Google Scholar
• Robertson WG, Peacock M, Hodgkinson A. Dietary changes and the incidence of urinary calculi in the U.K. between 1958 and 1976. J Chron Dis 1979; 32: 469–76
   PubMedGoogle Scholar
• Robertson WG, Peacock M, Heyburn PJ, Hanes FA, Rutherford A, Clementson E, et al. Should recurrent calcium oxalate stone formers become vegetarians?. Br J Urol 1979; 51: 427–31
   PubMedGoogle Scholar
• Robertson WG, Peacock M. The cause of idiopathic calcium stone disease: hypercalciuria or hyperoxaluria?. Nephron 1980; 26: 105–10
   PubMed Web of Science ®Google Scholar
• Robertson WG. Diet and calcium stones. Miner Electrolyte Metab 1987; 13: 228–34
   PubMedGoogle Scholar
• Lemann J. Composition of the diet and calcium kidney stones. N Engl J Med 1993; 328: 880–2
   PubMed Web of Science ®Google Scholar
• Lennon EJ, Lemann J, Relman AS. The effects of phosphoproteins on acid–base balance in normal subjects. J Clin Invest 1962; 41: 637–45
   PubMed Web of Science ®Google Scholar
• Kildeberg P. Quantitative acid-base physiology. System physiology and pathophysiology of renal, gastrointestinal, and skeletal acid-base metabolism. Igaku-Shoin Medical Publishers, Inc., New York 1981
   Google Scholar
• Osther PJ, Bollerslev J, Nørgaard JR, Engel K, Kildeberg P. Effects of acute acid loading on the risk of calcium phosphate and calcium oxalate crystallization in urine. Scanning Microsc 1994; 8: 63–9
   Google Scholar
• Cotlove E, Trantham HV, Bowman RL. An instrument and method for automatic, rapid, accurate, and sensitive titration of chloride in biologic samples. J Lab Clin Med 1967; 51: 461–8
   Google Scholar
• Baginski ES, Foá PP, Zak B. Microdetermination of inorganic phosphate, phospholipids, and total phosphate in biological materials. Clin Chem 1967; 13: 326–32
   PubMed Web of Science ®Google Scholar
• Yatzidas H, Oreopoulus D, Symvouludis A, Triantaphyllidis D, Yannitsiotis A. Une methode simple pour le dosage des sulfates dans le sérum et l’úrine. Revue Fr Etud Clin Biol 1964; 9: 126–7
   PubMedGoogle Scholar
• Lawrence R. Assay of serum inorganic phosphate without deproteinisation: automated and manual micromethods. Ann Clin Biochem 1974; 11: 234–40
   PubMed Web of Science ®Google Scholar
• Osther PJ, Engel K, Kildeberg P. Renal response to acute acid loading—an organ physiological approach. Scand J Urol Nephrol 2004; 38: 62–8
   PubMed Web of Science ®Google Scholar
• Kildeberg P. Acid-base status of biological fluids: amount of acid, kind of acid, anion-cation difference, and buffer value. Scand J Clin Lab Invest 1983; 43: 103–9
   PubMed Web of Science ®Google Scholar
• Pak CYC. Pathophysiology of calcium nephrolithiasis. In: Seldin DW, Giebisch G, editors. The kidney: physiology and pathophysiology. New York: Raven Press; p. 1365–79.
   Google Scholar
• McCurdy DK, Frederic M, Elkinton JR. Renal tubular acidosis due to amphotericin B. N Engl J Med 1968; 276: 124–30
   Google Scholar
• Battle DC, Kurtzmann NA. The defect in distal (Type 1) renal tubular acidosis. In: Gonick HC, Buckalew VM, editors. Renal tubular disorders. Pathophysiology, diagnosis, and management. New York: Marcel Dekker Inc.; 1985 p. 281–305.
   Google Scholar
• Bilogrov VM, Chugaj AV, Bessarabov VI. Urine pH variation dynamics in healthy individuals and stone formers. Urol Int 1990; 45: 326–31
   PubMed Web of Science ®Google Scholar
• Robertson WG, Peacock M. Review of risk factors in calcium oxalate urolithiasis. World J Urol 1983; 1: 114–8
   Web of Science ®Google Scholar
• Lemann J, Litzow JR, Lennon E. Studies of the mechanism by which chronic metabolic acidosis augments urinary calcium excretion in man. J Clin Invest 1967; 8: 1318–28
   Google Scholar
• Beck N, Webster SK. Effects of acute metabolic acidosis on parathyroid hormone action and calcium mobilization. Am J Physiol 1976; 230: 127–35
   PubMedGoogle Scholar
• Bushinsky DA, Levi-Setti R, Coe FL. Ion-microbe determination of bone surface elements: effects of a reduced medium pH. Am J Physiol 1986; 250: F1090–7
   PubMed Web of Science ®Google Scholar
• Lemann J, Jr, Lennon EJ, Goodman AD, Litzow JR, Relman AS. The net balance of acid in subjects given large loads of acid or alkali. J Clin Invest 1965; 44: 507–17
   PubMed Web of Science ®Google Scholar
• Coe FL, Firpo JJ, Hollandsworth DL, Segil L, Canterbury JM, Reiss E. Effect of acute and chronic metabolic acidosis on serum immunoactive parathyroid hormone in man. Kidney Int 1975; 8: 262–73
   Web of Science ®Google Scholar
• Green J, Kleeman R. Role of bone in regulation of systemic acid-base balance. Kidney Int 1991; 39: 9–26
   PubMed Web of Science ®Google Scholar
• Bushinsky DA, Lam BC, Nespeca R, Sessler NE, Grynpas MD. Decreased bone carbonate content in response to metabolic, but not respiratory, acidosis. Am J Physiol 1993; 265: F530–6
   PubMedGoogle Scholar
• Lennon EJ, Piering WF. A comparison of the effects of glucose ingestion and NH4Cl acidosis on urinary calcium and magnesium excretion in man. J Clin Invest 1970; 49: 1458–65
   PubMed Web of Science ®Google Scholar
• Adams ND, Gray RW, Leman J. The calciuria of increased fixed acid production in humans: evidence against a role for parathyroid hormone and 1,25(OH)2-vitamin D. Calcif Tissue Int 1979; 28: 233–8
   PubMed Web of Science ®Google Scholar
• Sutton RJ, Norman LM, Wong LM, Dirks JH. Effects of metabolic acidosis and alkalosis on sodium and calcium transport in the dog kidney. Kidney Int 1979; 15: 520–33
   PubMed Web of Science ®Google Scholar
• Knispel HH, Fitzner R, Kaiser M, Butz M. Acute acid load in recurrent stone-formers. Urol Int 1988; 43: 93–6
   PubMed Web of Science ®Google Scholar
• Osther PJ, Bollerslev J, Hansen AB, Engel K, Kildeberg P. On the calciuric response to acid loading. Urol Res 1993; 21: 153
   Google Scholar
• Malluche HH, Tschope W, Ritz E, Meyer-Sabellek, Massry SG. Abnormal bone histology in idiopathic hypercalciuria. J Clin Endocrinol Metab 1980; 50: 654–8
   PubMed Web of Science ®Google Scholar
• Steinicke T, Mosekilde L, Christensen MS, Melsen F. A histomorphometric determination of iliac bone remodelling in patients with recurrent renal stone formation and idiopathic hypercalciuria. APMIS 1989; 97: 309–16
   PubMed Web of Science ®Google Scholar
• Bataille P, Achard JM, Fournier A, Boudailliez B, Wasteel PF, Esper NE, et al. Diet, vitamin D and vertebral mineral density in hypercalciuric calcium stone formers. Kidney Int 1991; 39: 1193–205
   PubMed Web of Science ®Google Scholar
• Pietschmann F, Breslau NA, Pak CYC. Reduced vertebral bone density in hypercalciuric nephrolithiasis. J Bone Miner Res 1992; 7: 1383–8
   PubMed Web of Science ®Google Scholar
• Fuss M, Pepersack T, Van Geel J, Corvilain J, Vandewalle J-C, Bergman P, et al. Involvement of low-calcium diet in the reduced bone mineral content of idiopathic renal stone formers. Calcif Tissue Int 1990; 46: 9–13
   PubMed Web of Science ®Google Scholar