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Short Communication

A low rate of factor V Leiden mutation among Sudanese women with deep venous thrombosis during pregnancy and puerperium

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References

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  • Abdulkadir J, Feleke Y, Berg JP, Falch JA, Odegaard OR. 1997. Absence of the factor V Leiden mutation in Ethiopians. Thrombosis Research 86:431–432.
  • Abu-Skeen IA, Mohamed AA, Moustafa NN, Badawy ME. 2010. Factor V Leiden and prothrombin G20210A gene mutations in women with a history of thrombosis during pregnancy. Relation to pregnancy outcomes for mother and fetus. Saudi Medical Journal 31:123–129.
  • Almawi WY, Tamim H, Kreidy R, Timson G, Rahal E, Nabulsi M, et al. 2005. A case control study on the contribution of factor V-Leiden, prothrombin G20210A, and MTHFR C677T mutations to the genetic susceptibility of deep venous thrombosis. Journal of Thrombosis and Thrombolysis 19:189–196.
  • Doyle NM, Monga M. 2004. Thromboembolic disease in pregnancy. Obstetrics and Gynecology Clinics of North America 31:319–344.
  • Haggaz AA, Mirghani OA, Adam I. 2003. Venous thromboembolism in pregnancy and the puerperium in Sudanese women. International Journal of Gynaecology and Obstetrics 83:309–310.
  • Jadaon MM. 2011. Epidemiology of activated protein C resistance and factor V Leiden mutation in the Mediterranean region. Mediterranean Journal of Hematology and Infectious Disease 3:e2011037.

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