Assessment of oxidative/anti-oxidative markers and DNA damage profile induced by chemotherapy in algerian children with lymphoma

References

• Bartlett, J.J., et al., 2016. Doxorubicin impairs cardiomyocyte viability by suppressing transcription factor EB expression and disrupting autophagy. Biochemical Journal, 473(21), 3769–3789.
   PubMed Web of Science ®Google Scholar
• Bolzán, A.D., and Bianchi, M.S., 2018. DNA and chromosome damage induced by bleomycin in mammalian cells: An update. Mutation Research/Reviews in Mutation Research, 775, 51–62.
   PubMed Web of Science ®Google Scholar
• Cannan, W.J., et al., 2014. Nucleosomes suppress the formation of double-strand DNA breaks during attempted base excision repair of clustered oxidative damages. Journal of Biological Chemistry, 289(29), 19881–19893.
   PubMed Web of Science ®Google Scholar
• Cheuk, D.K., et al., 2016. Medical interventions for treating anthracycline-induced symptomatic and asymptomatic cardiotoxicity during and after treatment for childhood cancer. Cochrane Database of Systematic Reviews, 23(8), CD008011.
   Google Scholar
• Conklin, K.A., 2004. Chemotherapy-associated oxidative stress: impact on chemotherapeutic effectiveness. Integrative Cancer Therapies, 3(4), 294–300.
   PubMedGoogle Scholar
• Deavall, D.G., et al., 2012. Drug-Induced Oxidative Stress and Toxicity. Journal of Toxicology, 13, 645460.
   Google Scholar
• El-Mezayen, H.A., et al., 2015. Oxidant/antioxidant status and their relations to chemotherapy in non-Hodgkin’s lymphoma. International Journal of Pharmaceutical and Clinical Research, 7(4), 269–274.
   Google Scholar
• Figueroa-Gonzalez, G., and Perez-Plasencia, C., 2017. Strategies for the evaluation of DNA damage and repair mechanisms in cancer. Oncology Letters, 13, 3982–3988.
   PubMed Web of Science ®Google Scholar
• Gadjeva, V., Kuchukova, D., and Georgieva, R., 2005. Influence of poly-chemotherapy on the antioxidant levels and lipid peroxidation in patients with lymphoproliferative disease. Comparative Clinical Pathology, 14(1), 13–18.
   Google Scholar
• Imbesi, S., et al., 2013. Oxidative stress in oncohematologic diseases: An update. Expert Review of Hematology, 6(3), 317–325.
   PubMed Web of Science ®Google Scholar
• Kaya, E., et al., 2005. Oxidant/antioxidant parameters and their relationship with chemotherapy in Hodgkin's lymphoma. Journal of International Medical Research, 33(6), 687–692.
   PubMed Web of Science ®Google Scholar
• Kudryavtseva, A.V., et al., 2016. Mitochondrial dysfunction and oxidative stress in aging and cancer. Oncotarget, 7(29), 4879–44905.
   Google Scholar
• Kvaerner, A.S., et al., 2018. DNA damage in blood cells in relation to chemotherapy and nutritional status in colorectal cancer patients-A pilot study. DNA Repair, 63, 16–24.
   PubMed Web of Science ®Google Scholar
• Marklund, S., and Marklund, G., 1974. Involvement of the superoxide anion radical in the autoxidation of pyrogallol and a convenient assay for superoxide dismutase. European Journal of Biochemistry, 47(3), 469–474.
   PubMedGoogle Scholar
• Marullo, R., et al., 2013. Cisplatin induces a mitochondrial-ROS response that contributes to cytotoxicity depending on mitochondrial redox status and bioenergetic functions. PLoS One, 8(11), e81162.
   PubMed Web of Science ®Google Scholar
• Mesquita, C.S., et al., 2014. Simplified 2,4-dinitrophenylhydrazine spectrophotometric assay for quantification of carbonyls in oxidized proteins. Analytical Biochemistry, 458, 69–71.
   PubMed Web of Science ®Google Scholar
• Morabito, F., et al., 2004. Lipid peroxidation and protein oxidation in patients affected by Hodgkin's lymphoma. Mediators of Inflammation, 13(5–6), 381–383.
   PubMed Web of Science ®Google Scholar
• Ohkawa, H., Ohishi, N., and Yagi, K., 1979. Assay for lipid peroxides in animal tissues by thiobarbituric acid reaction. Analytical Biochemistry, 95(2), 351–358.
   PubMed Web of Science ®Google Scholar
• Piegari, E., et al., 2016. MicroRNA-34a regulates doxorubicin-induced cardiotoxicity in rat. Oncotarget, 7(38), 62312–62326.
   PubMedGoogle Scholar
• Popadiuk, S., et al., 1998. Carbonyl groups content on the basis of protein peroxidation analysis with total antioxidant status in blood of children with cancers. Wiadomości Lekarskie, 5, 107–112.
   Google Scholar
• Salehi, F., et al., 2018. Oxidative DNA damage induced by ROS-modulating agents with the ability to target DNA: a comparison of the biological characteristics of citrus pectin and apple pectin. Science Report, 8(1), 13902.
   PubMed Web of Science ®Google Scholar
• Singh, N.P., et al., 1988. A simple technique for quantitation of low levels of DNA damage in individual cells. Experimental Cell Research, 175(1), 184–191.
   PubMed Web of Science ®Google Scholar
• Singh, R.K., et al., 2009. Studies on biomarkers for oxidative stress in patients with chronic myeloid leukemia. Hematology/Oncology and Stem Cell Therapy, 2(1), 285–288.
   PubMedGoogle Scholar
• Steliarova-Foucher, E., et al., 2017. International incidence of childhood cancer, 2001–2010: a population-based registry study. Lancet Oncol, 18(6), 719–731.
   PubMed Web of Science ®Google Scholar
• Swerdlow, S.H., et al., 2016. The 2016 revision of the World Health Organization classifcation of lymphoid neoplasms. Blood, 127(20), 2375–2390.
   PubMed Web of Science ®Google Scholar
• Yu, W., et al., 2018. Cisplatin generates oxidative stress which is accompanied by rapid shifts in central carbon metabolism. Scientific Reports, 8(1), 4306.
   PubMed Web of Science ®Google Scholar
• Zhang, J., et al., 2018. Oxidative stress response induced by chemotherapy in leukemia treatment. Molecular and Clinical Oncology, 8, 391–399.
   PubMed Web of Science ®Google Scholar