References
- Jamshidi J, Movafagh A, Emamalizadeh B, et al. HLA-DRA is associated with Parkinson’s disease in Iranian population. Int J Immunogenet. 2014;41(6):508–511.10.1111/iji.2014.41.issue-6
- MacLeod DA, Rhinn H, Kuwahara T, et al. RAB7L1 interacts with LRRK2 to modify intraneuronal protein sorting and parkinson’s disease risk. Neuron. 2013;77(3):425–439.10.1016/j.neuron.2012.11.033
- Emamalizadeh B, Movafagh A, Akbari M, et al. RIT2, a susceptibility gene for Parkinson’s disease in Iranian population. Neurobiol Aging. 2014;35(12):e27–e28.10.1016/j.neurobiolaging.2014.07.013
- Gao J, Xu H, Huang X, et al. Short communication: genetic variations of SLC2A9 in relation to Parkinson’s disease. Transl Neurodegener. 2013;2(1):5.
- Wood-Kaczmar A, Gandhi S, Wood N. Understanding the molecular causes of Parkinson’s disease. Trends Mol Med. 2006;12(11):521–528.10.1016/j.molmed.2006.09.007
- Gan-Or Z, Bar-Shira A, Dahary D, et al. Association of sequence alterations in the putative promoter of RAB7L1 with a reduced parkinson disease risk. Arch Neurol. 2012;69(1):105–110.10.1001/archneurol.2011.924
- Shimizu F, Katagiri T, Suzuki M, et al. Cloning and chromosome assignment to 1q32 of a human cDNA (RAB7L1) encoding a small GTP-binding protein, a member of the RAS superfamily. Cytogenet Genome Res. 1997;77(3–4):261–263.10.1159/000134591
- De Lau LM, Breteler MM. Epidemiology of Parkinson’s disease. Lancet Neurol. 2006;5(6):525–535.10.1016/S1474-4422(06)70471-9
- Satake W, Nakabayashi Y, Mizuta I, et al. Genome-wide association study identifies common variants at four loci as genetic risk factors for Parkinson’s disease. Nat Genet. 2009;41(12):1303–1307.10.1038/ng.485
- Simón-Sánchez J, Schulte C, Bras JM, et al. Genome-wide association study reveals genetic risk underlying Parkinson’s disease. Nat Genet. 2009;41(12):1308–1312.10.1038/ng.487
- Tan E-K, Kwok H-K, Tan L, et al. Analysis of GWAS-linked loci in Parkinson disease reaffirms PARK16 as a susceptibility locus. Neurology. 2010;75(6):508–512.10.1212/WNL.0b013e3181eccfcd
- Tucci A, Nalls MA, Houlden H, et al. Genetic variability at the PARK16 locus. Eur J Hum Genet. 2010;18(12):1356–1359.10.1038/ejhg.2010.125
- Xia H, Luo Q, Li XX, et al. Association between PARK16 gene polymorphisms and susceptibility of Parkinson’s disease in a Chinese population. Genet Mol Res. 2015;14(2):2978–2985.10.4238/2015.April.10.7
- Mata IF, Yearout D, Alvarez V, et al. Replication of MAPT and SNCA, but not PARK16-18, as susceptibility genes for Parkinson’s disease. Mov Disord. 2011;26(5):819–823.10.1002/mds.v26.5
- Consortium IPDG. Imputation of sequence variants for identification of genetic risks for Parkinson’s disease: a meta-analysis of genome-wide association studies. Lancet. 2011;377(9766):641–649.
- Goudarzian M, Khaligh A, Fourozan R, et al. The rs1572931 polymorphism of the RAB7L1 gene promoter is associated with reduced risk of Parkinson’s disease. Neurol Res. 2015;37(11):1029–1031. PubMed PMID: 26344175.10.1179/1743132815Y.0000000085