References
- Coutinho M., Gerstein H. C., Wang Y., Yusuf S. The relationship between glucose and incident cardiovascular events. A metaregression analysis of published data from 20 studies of 95,783 individuals followed for 12.4 years. Diabetes Care 1999; 22: 233–40
- Benetos A., Safar M., Rudnichi A., Smulyan H., Richard J‐L. Pulse pressure: A predictor of longterm cardiovascular mortality in a French male population. Hypertension 1997; 30: 1410–5
- Franklin S. S., Khan S. A., Wong N. D., Larson M. G., Levy D. Is pulse pressure useful in predicting risk for coronary heart disease? The Framingham Heart Study. Circulation 1999; 100: 354–60
- Jankowski P., Kawecka‐Jaszcz K., Czarnecka D., Brzozowska‐Kiszka M., Styczkiewicz K., Styczkiewicz M., et al. Ascending aortic, but not brachial blood pressure‐derived indices are related to coronary atherosclerosis. Atherosclerosis 2004; 176: 151–5
- Nishijima T., Nakayama Y., Tsumura K., Yamashita N., Yoshimaru K., Ueda H., et al. Pulsatility of ascending aortic blood pressure waveform is associated with an increased risk of coronary heart disease. Am J Hypertens 2001; 14: 469–73
- Guray Y., Guray U., Altay H., Cay S., Yilmaz M. B., Kisacik H. L., et al. Aortic pulse pressure and aortic pulsatility are associated with angiographic coronary artery disease in women. Blood Press 2005; 14: 293–7
- Wakabayashi I., Masuda H. Association of pulse pressure with carotid atherosclerosis in patients with type 2 diabetes mellitus. Blood Press 2007; 16: 56–62
- Gibson C. M., Cannon C. P., Daley W. L., Dodge J. T Jr., Alexander B Jr., Marble S. J., et al. TIMI frame count: A quantitative method of assessing coronary artery flow. Circulation 1996; 93: 879–88
- Friedewald W. T., Levy R. I., Fredrickson D. S. Estimation of the concentration of low density lipoprotein cholesterol in plasma, without use of the preparative ultracentrifuge. Clin Chem 1972; 18: 499–502
- Weber T., Auer J., O'Rourke M. F., Kvas E., Lassnig E., Berent R., et al. Arterial stiffness, wave reflections, and the risk of coronary artery disease. Circulation 2004; 109: 184–9
- Furchgott R. F., Zawadzki J. V. The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine. Nature 1980; 288: 373–6
- Moncada S., Higgs A. The L‐arginine‐nitric oxide pathway. N Engl J Med 1993; 329: 2002–12
- McEniery C. M., Wallace S., Mackenzie I. S., McDonnell B., Yasmin., Newby D. E., et al. Endothelial function is associated with pulse pressure, pulse wave velocity, and augmentation index in healthy humans. Hypertension 2006; 48: 602–8
- Binak E., Gunduz H., Sahin M., Kurtoglu N., Dindar I. The relation between impaired glucose tolerance and slow coronary flow. Int J Cardiol 2006; 111: 142–6
- Caballero A. E., Arora S., Saouaf R., Lim S. C., Smakowski P., Park J. Y., et al. Microvascular and macrovascular reactivity is reduced in subjects at risk for type 2 diabetes. Diabetes 1999; 48: 1856–62
- Srinivasan M., Herrero P., McGill J. B., Bennik J., Heere B., Lesniak D., et al. The effects of plasma insulin and glucose on myocardial blood flow in patients with type 1 diabetes mellitus. J Am Coll Cardiol 2005; 46: 42–8
- Tesfamariam B., Brown M. L., Deykin D., Cohen R. A. Elevated glucose promotes generation of endothelium‐derived vasoconstrictor prostanoids in rabbit aorta. J Clin Invest 1990; 85: 929–32
- Fuller J. H., Shipley M. J., Rose G., Jarrett R. J., Keen H. Coronary heart disease risk and impaired glucose tolerance: The Whitehall study. Lancet 1980; 1: 1373–6
- Bucala R., Tracey K. J., Cerami A. Advanced glycosylation products quench nitric oxide and mediate defective endothelium‐dependent vasodilatation in experimental diabetes. J Clin Invest 1991; 87: 432–8
- De Angelis L., Marfella M. A., Siniscalchi M., Marino L., Nappo F., Giugliano F., et al. Erectile and endothelial dysfunction in Type II diabetes: A possible link. Diabetologia 2001; 44: 1155–60