REFERENCES
- Rivera F, López-Gómez JM, Pérez-García R, on the behalf of the Spanish Registry of Glomerulonephritis. Clinicopathologic correlations of renal pathology in Spain. Kidney Int. 2004; 66: 898–904
- Bonomini V, Stefoni S, Vangelista A. Long-term patient and renal prognosis in acute kidney injury. Nephron. 1984; 36: 169–172
- Abdulkader R, Malheiro P, Daher E, et al. Late evaluation of glomerular filtration rate, proteinuria, and urinary acidification after acute tubular necrosis. Ren Fail. 1992; 14: 57–61
- Finn WF. Recovery from acute renal failure. Acute Renal Failure, JM Lazarus, BM Brenner. Churchill Livingstone, New York 1993; 553–596
- Basile DP, Donohoe D, Roethe K, Osborn JL. Renal ischemic injury results in permanent damage to peritubular capillaries and influences long-term function. Am J Physiol Ren Physiol. 2001; 281: F887–F899
- Forbes JM, Hewitson TD, Becker GJ, Jones CL. Ischemic acute kidney injury: Long-term histology of cell and matrix changes in the rat. Kidney Int. 2000; 57: 2375–2385
- Soares TJ, Costa RS, Volpini RA, da Silva CG, Coimbra TM. Long-term evolution of the acute tubular necrosis (ATN) induced by glycerol: Role of myoblasts and macrophages. Int J Exp Pathol. 2002; 83: 165–172
- Striker GE, Quadracci LJ, Cutler RE. Use and interpretation of renal biopsy. Major Problems in Pathology, JL Bennington. W.B. Saunders, Philadelphia 1978; 1–347
- Moyses-Neto M, Costa RS, Volpini RA, Garcia TMP, Rodrigues FF, Coimbra TM. Interstitial alterations in renal cortex in acute tubular necrosis (ATN) post-renal transplantation and in patients with ATN not related to renal transplantation. Clin Transplant. 2004; 18: 156–165
- Yang L, Li X, Wang H. Possible mechanisms explaining the tendency towards interstitial fibrosis in aristolochic acid-induced acute tubular necrosis. Nephrol Dial Transplant. 2007; 22: 445–456
- National Kidney Foundation. K/DOQI clinical practice guidelines for chronic kidney disease: Evaluation, classification, and stratification. Am J Kidney Dis. 2002; 39(Suppl. 1)S1–S266
- O'Valle F, Benitez MC, Gómez-Morales M, et al. Correlation of morphological findings with functional reserve in aging donor: Role of poly(ADP-ribose) polymerase. Transplant Proc. 2004; 36: 733–735
- Gutiérrez E, González E, Hernández E, et al. Factors that determine an incomplete recovery of renal function in macrohematuria-induced acute renal failure of IgA nephropathy. Clin J Am Soc Nephrol. 2007; 2: 51–57
- Pagtalunan ME, Olson JL, Tilney NL, Meyer TW. Late consequences of acute ischemic injury to a solitary kidney. J Am Soc Nephrol. 1999; 10: 366–373
- Geleilete TJ, Melo GC, Costa RS, Volpini RA, Soares TJ, Coimbra TM. Role of myoblasts, macrophages, transforming growth factor-beta, endothelin, angiotensin II, and fibronectin in the progression of tubulointerstitial nephritis induced by gentamicin. J Nephrol. 2002; 15: 633–642
- Chkhotua A, Shapira Z, Tovar A, Shabtai E, Yussim A. Cellular senescence: A new marker of kidney function recovery after ischemic injury in rats. Transplant Proc. 2001; 33: 2910–2915
- Schiffl H. Renal recovery from acute tubular necrosis requiring renal replacement therapy: A prospective study in critically ill patients. Nephrol Dial Transplant. 2006; 21: 1248–1252
- Bell M, NG SWI, Granath F, Schon S, Ekbom A, Martling CR. Continuous renal replacement therapy is associated with less chronic renal failure than intermittent haemodialysis after acute renal failure. Intensive Care Med. 2007; 33: 773–780
- Ali T, Khan I, Simpson W, et al. Incidence and outcomes in acute kidney injury: A comprehensive population-based study. J Am Soc Nephrol. 2007; 18: 1292–1298