References
- Jahns R, Boivin V, Siegmund C, Inselmann G, Lohse MJ, Boege F. Autoantibodies activating human β1-adrenergic receptors are associated with reduced cardiac function in chronic heart failure. Circulation 1999; 99: 649–654
- Iwata M, Yoshikawa T, Baba A, Anzai T, Mitamura H, Ogawa S. Autoantibodies against the second extracellular loop of beta1-adrenergic receptors predict ventricular tachycardia and sudden death in patients with idiopathic dilated cardiomyopathy. J Am Coll Cardiol 2001; 37: 418–424
- Wallukat G, Wollenberger A, Morwinski R, Pitschner HF. Anti-β1-adrenoceptor autoantibodies with chronotropic activity from the serum of patients with dilated cardiomyopathy: Mapping of epitopes in the first and second extracellular loops. J Mol Cell Cardiol 1995; 27: 397–406
- Staudt Y, Mobini R, Fu M, Felix SB, Kühn JP, Staudt A. β1-adrenoceptor antibodies induce apoptosis in adult isolated cardiomyocytes. Eur J Pharmacol 2003; 466: 1–6
- Staudt A, Staudt Y, Dörr M, Böhm M, Knebel F, Hummel A, Wunderle L, Tiburcy M, Wernecke KD, Baumann G, Felix SB. Potential role of humoral immunity in cardiac dysfunction of patients suffering from dilated cardiomyopathy. J Am Coll Cardiol 2004; 44: 829–836
- Jahns R, Boivin V, Hein L, Triebel S, Angermann CE, Ertl G, Lohse MJ. Direct evidence for a β1-adrenergic receptor-directed autoimmune attack as a cause of idiopathic dilated cardiomyopathy. J Clin Invest 2004; 113: 1419–1429
- Liu J, Mao W, Iwai C, Fukuoka S, Vulapalli R, Huang H, Sharma V, Sheu S-S, Fu M, Liang C-S. Adoptive passive transfer of rabbit β-adrenoceptor peptide immune cardiomyopathy into the Rag2− / − mouse: Participation of the ER stress. J Mol Cell Cardiol 2008; 44: 304–314
- Matsui S, Larsson L, Hayase M, Katsuda S, Teraoka K, Kurihara T, Murano H, Nishikawa K, Fu M. Specific removal of β1-adrenoceptor autoantibodies by immunoabsorption in rabbits with autoimmue cardiomyopathy improved cardiac structure and function. J Mol Cell Cardiol 2006; 41: 78–85
- Wallukat G, Muller J, Hetzer R. Specific removal of β1-adrenergic antibodies from patients with idiopathic dilated cardiomyopathy. N Engl J Med 2002; 347: 1806
- Mao W, Fukuoka S, Iwai C, Liu J, Sharma VL, Sheu SS, Fu M, Liang C-S. Cardiomyocyte apoptosis in autoimmune cardiomyopathy: Mediated via endoplasmic reticulum stress and exaggerated by norepinephrine. Am J Physiol Heart Circ Physiol 2007; 293: H1636–H1645
- Lamkanfi M, Kalai M, Vandenabeele P. Caspase-12: An overview. Cell Death Differ 2004; 11: 365–368
- Morishima N, Nakanishi K, Takenouchi H, Shibata T, Yasuhiko Y. An endoplasmic reticulum stress-specific caspase cascade in apoptosis. Cytochrome c-independent activation of caspase-9 by caspase-12. J Biol Chem 2002; 277: 34287–34294
- Liu H, Bowes RC, III, van de Water B, Sillence C, Nagelkerke JF, Stevens JL. Endoplasmic reticulum chaperone GRP78 and calreticulin prevent oxidative stress, Ca2+ disturbance, and cell death in renal epithelial cells. J Biol Chem 1997; 272: 21751–21759
- Rao RV, Hermel E, Castro-Obregon S, del Rio G, Ellerby LM, Ellerby HM, Bredesen DE. Coupling endoplasmic reticulum stress to the cell death program. Mechanism of caspase activation. J Biol Chem 2001; 276: 33869–33874
- Rao RV, Peel A, Logvinova A, del Rio G, Hermel E, Yokota T, Goldsmith PC, Ellerby LM, Ellerby HM, Bredesen DE. Coupling endoplasmic reticulum stress to the cell death program: Role of the ER chaperone GRP78. FEBS Lett 2002; 514: 122–128
- Takeda K, Matsuzawa A, Nishitoh H, Tobiume K, Kishida S, Ninomiya Tsuji J, Matsumoto K, Ichijo H. Involvement of ASK1 in Ca2+ induced p38 MAP kinase activation. EMBO Rep 2004; 5: 161–166
- Staudt A, Eichler P, Trimpert C, Felix SB, Greinacher A. Fcγ receptors IIa on cardiomyocytes and their potential functional relevance in dilated cardiomyopathy. J Am Coll Cardiol 2007, 49: 1684–1692
- Mao W, Iwai C, Keng P, Vulapalli R, Liang C-S. NE-induced oxidative stress causes PC12 cell apoptosis by both ER stress and mitochondrial intrinsic pathway: Inhibition of the phosphatidylinositol 3-kinase survival pathway. Am J Physiol Cell Physiol 2006; 290: C1373–C1384
- Srinivasan S, Ohsugi M, Liu Z, Fatrai S, Bernaal-Mizrachi E, Permutt MA. Endoplasmic reticulum stress-induced apoptosis is partly mediated by reduced insulin signaling through phosphatidylinositol 3-kinase/Akt and increased glycogen synthase kinase-3β in mouse insulinoma cells. Diabetes 2005; 54: 968–975
- Hyoda K, Hosoi T, Horie N, Okuma Y, Ozawa K, Nomura Y. PI3K-Akt inactivation induced CHOP expression in endoplasmic reticulum-stressed cells. Biochem Biophys Res Commun 2006; 340: 286–290