References
- Fuster V, Badimon L, Badimon JJ, Chesebro JH. The pathogenesis of coronary artery disease and the acute coronary syndromes (1) and (2). New Engl J Med 1992; 326: 242, 310–250, 318
- Libby P. Current concept of the pathogenesis of the acute coronary syndromes. Circulation 2001; 104: 365–372
- Mulvihill NT, Foley JB. Inflammation in acute coronary syndromes. Heart 2002; 87: 201–204
- Beck B, Weintraub W, Alexander R. Elevation of C-reactive protein in ‘active’ coronary artery disease. Am J Cardiol 1990; 65: 168–172
- World Health Organization. Report of the Joint International Society and Federation of Cardiology/World Health Organization Task Force on Standardization of Clinical Nomenclature. Nomenclature and criteria for diagnosis of ischemic heart disease. Circulation 1979; 59: 607–609
- Lanza GA, Andreotti F, Sestito A, Sciahbasi A, Crea F, Maseri A. Platelet aggregability in cardiac syndrome X. Eur Heart J 2001; 22: 1924–1930
- Lanza GA, Sestito A, Iacovella S, Morlacchi L, Romagnoli E, Schiavoni G, Crea F, Maseri A, Andreotti F. Relation between platelet response to exercise and coronary angiographic findings in patients with effort angina. Circulation 2003; 107: 1378–1382
- Sestito A, Sciahbasi A, Landolfi R, Maseri A, Lanza GA, Andreotti F. A simple assay for platelet-mediated hemostasis in flowing whole blood (PFA-100): reproducibility and effects of sex and age. Cardiologia 1999; 44: 661–665
- Marshall PW, Williams AJ, Dixon RM, Growcott JW, Warburton S, Armstrong J, Moores J. A comparison of the effects of aspirin on bleeding time measured using the Simplate method and closure time measured using the PFA-100, in healthy volunteers. Br J Clin Pharmacol 1997; 44: 151–155
- Homoncik M, Jilma B, Hergovich N, Stohlawetz P, Panzer S, Speiser W. Monitoring of aspirin (ASA) pharmacodynamics with the platelet function analyzer PFA-100. Thromb Haemost 2000; 83: 316–321
- Sobel M, Salzman EW, Davies GC, Handin RI, Sweeney J, Ploetz J, Kurland G. Circulating platelet products in unstable angina pectoris. Circulation 1981; 63: 300–306
- Fitzgerald DJ, Roy L, Catella F, Fitzgerald GA. Platelet activation in unstable coronary disease. New Engl J Med 1986; 315: 983–989
- Eto K, Takeshita S, Ochiai M, Ozaki Y, Sato T, Isshiki T. Platelet aggregation in acute coronary syndromes: Use of a new aggregometer with laser light scattering to assess platelet aggregability. Cardiovasc Res 1998; 40: 223–229
- Kajiwara I, Ogawa H, Soejima H, Takazoe K, Miyamoto S, Sakamoto T, Yoshimura M, Kugiyama K, Yasue H. The prognostic value of small-sized platelet aggregates in unstable angina: Detection by a novel laser-light scattering method. Thromb Res 2001; 101: 109–118
- Miyamoto S, Ogawa H, Sakamoto T, Soejima H, Takazoe K, Shimomura H, Kajiwara I, Yoshimura M, Kugiyama K, Nakamura S, Ozaki Y. Platelet hyperaggregability persists even after the improvement of increased blood coagulation and impaired fibrinolysis with the stabilization of symptoms in patients with unstable angina. Int J Cardiol 2001; 80: 235–242
- Liuzzo G, Biasucci LM, Gallimore JR, Grillo RL, Rebuzzi AG, Pepys MB, Maseri A. The prognostic value of C-reactive protein and serum amyloid A protein in severe unstable angina. New Engl J Med 1994; 331: 417–424
- Biasucci LM, Liuzzo G, Grillo RL, Caligiuri G, Rebuzzi AG, Buffon A, Summaria F, Ginetti F, Fadda G, Maseri A. Elevated levels of C-reactive protein at discharge in patients with unstable angina predict recurrent instability. Circulation 1999; 99: 855–860
- Fiedel BA, Gewurz H. Effects of C-reactive protein on platelet function. I. Inhibition of platelet aggregation and release reactions. J Immunol 1976; 116: 1289–1294
- Fiedel BA, Gewurz H. Effects of C-reactive protein on platelet function. II. Inhibition by CRP of platelet reactivities stimulated by poly-L-lysine, ADP, epinephrine, and collagen. J Immunol 1976; 117: 1073–1078
- Miyazawa K, Kiyono S, Inoue K. Modulation of stimulus-dependent human platelet activation by C-reactive protein modified with active oxygen species. J Immunol 1988; 141: 570–574