The Candida albicans mating type like locus [MTL] is not involved in chlamydospore formation

References

• Kwon-Chung KJ, Bennett JE. Medical Mycology. Lea & Febiger, Philadelphia 1992
   Google Scholar
• Odds FC. Candida and Candidosis: a Review and Bibliography. Bailliere Tindall, London 1988
   Google Scholar
• Raudonis BM, Smith AG. Germination of the chlamydospores of Candida albicans. Mycopathologia 1982; 78: 87–91
   PubMed Web of Science ®Google Scholar
• Jansons VK, Nickerson WJ. Induction, morphogenesis, and germination of the chlamydospore of Candida albicans. J Bacteriol 1970; 104: 910–921
   PubMed Web of Science ®Google Scholar
• McGinnis MR. Laboratory handbook of medical mycology. Academic Press, New York 1980
   Google Scholar
• Gutierrez J, Morales P, Gonzalez MA, et al. Candida dubliniensis, a new fungal pathogen. J Basic Microbiol 2002; 42: 207–227
   PubMed Web of Science ®Google Scholar
• Staib F, Arasteh K. Chlamydospore formation on Staib agar. Observations made before Candida dubliniensis was described. Mycoses 2001; 44: 23–27
   PubMed Web of Science ®Google Scholar
• Staib P, Morschhauser J. Liquid growth conditions for abundant chlamydospore formation in Candida dubliniensis. Mycoses 2005; 48: 50–54
   PubMed Web of Science ®Google Scholar
• Jabra RMA, Baqui AAMA, Kelley JI, et al. Identification of Candida dubliniensis in a prospective study of patients in the United States. J Clin Microbiol 1999; 37: 321–326
   PubMed Web of Science ®Google Scholar
• Staib P, Morschhauser J. Chlamydospore formation on Staib agar as a species-specific characteristic of Candida dubliniensis. Mycoses 1999; 42: 521–524
   PubMed Web of Science ®Google Scholar
• Cole GT, Seshan KR, Phaneuf M, et al. Chlamydospore-like cells of Candida albicans in the gastrointestinal tract of infected, immunocompromised mice. Can J Microbiol 1991; 37: 637–646
   PubMed Web of Science ®Google Scholar
• Chabasse D, Bouchara JP, de Gentile L, et al. Candida albicans chlamydospores observed in vivo in a patient with AIDS. Ann Biol Clin 1988; 46: 817–818
   PubMed Web of Science ®Google Scholar
• Mitchell AP, Herskowitz I. Activation of meiosis and sporulation by repression of the RME1 product in yeast. Nature 1986; 319: 738–742
   PubMed Web of Science ®Google Scholar
• Sonneborn A, Bockmuhl DP, Ernst JF. Chlamydospore formation in Candida albicans requires the Efg1p morphogenetic regulator. Infect Immun 1999; 67: 5514–5517
   PubMed Web of Science ®Google Scholar
• Staib P, Morschhauser J. Differential expression of the NRG1 repressor controls species-specific regulation of chlamydospore development in Candida albicans and Candida dubliniensis. Mol Microbiol 2005; 55: 637–652
   PubMed Web of Science ®Google Scholar
• Richard M, Ibata OS, Dromer F, et al. Complete glycosylphosphatidylinositol anchors are required in Candida albicans for full morphogenesis, virulence and resistance to macrophages. Mol Microbiol 2002; 44: 841–853
   Google Scholar
• Martin SW, Douglas LM, Konopka JB. Cell cycle dynamics and quorum sensing in Candida albicans chlamydospores are distinct from budding and hyphal growth. Eukaryot Cell 2005; 4: 1191–1202
   PubMedGoogle Scholar
• Alonso MR, Navarro GF, Molero G, et al. Role of the mitogen-activated protein kinase hog1p in morphogenesis and virulence of Candida albicans. J Bacteriol 1999; 181: 3058–3068
   PubMed Web of Science ®Google Scholar
• Alonso MR, Navarro GF, Roman E, et al. The Hog1 mitogen-activated protein kinase is essential in the oxidative stress response and chlamydospore formation in Candida albicans. Eukaryot Cell 2003; 2: 351–361
   PubMedGoogle Scholar
• Nobile CJ, Bruno VM, Richard ML, et al. Genetic control of chlamydospore formation in Candida albicans. Microbiology 2003; 12: 3629–3637
   Google Scholar
• Tzung KW, Williams RM, Scherer S, et al. Genomic evidence for a complete sexual cycle in Candida albicans. Proc Natl Acad Sci USA 2001; 98: 3249–3253
   PubMed Web of Science ®Google Scholar
• Hull CM, Johnson AD. Identification of a mating type-like locus in the asexual pathogenic yeast Candida albicans. Science 1999; 285: 1271–1275
   PubMed Web of Science ®Google Scholar
• Lockhart SR, Pujol C, Daniels KJ, et al. In Candida albicans, white-opaque switchers are homozygous for mating type. Genetics 2002; 162: 737–745
   PubMedGoogle Scholar
• Bennett RJ, Johnson AD. Completion of a parasexual cycle in Candida albicans by induced chromosome loss in tetraploid strains. EMBO J 2003; 22: 2505–2515
   PubMed Web of Science ®Google Scholar
• Hull CM, Raisner RM, Johnson AD. Evidence for mating of the ‘asexual’ yeast Candida albicans in a mammalian host. Science 2000; 289: 307–310
   PubMed Web of Science ®Google Scholar
• Magee BB, Magee PT. Induction of mating in Candida albicans by construction of MTL a and MTL alpha strains. Science 2000; 289: 310–313
   PubMed Web of Science ®Google Scholar
• Rustad TR, Stevens DA, Pfaller MA, et al. Homozygosity at the Candida albicans MTL locus associated with azole resistance. Microbiology 2002; 148: 1061–1072
   PubMed Web of Science ®Google Scholar
• Fonzi WA, Irwin MY. Isogenic strain construction and gene mapping in Candida albicans. Genetics 1993; 134: 717–728
   PubMed Web of Science ®Google Scholar
• Marr KA, Lyons CN, Rustad TR, et al. Rapid, transient fluconazole resistance in Candida albicans is associated with increased mRNA levels of CDR. Antimicrob Agents Chemother 1998; 42: 2584–2589
   PubMed Web of Science ®Google Scholar
• White TC, Pfaller MA, Rinaldi RG, et al. Stable azole drug resistance associated with a substrain of Candida albicans from an HIV-infected patient. Oral Dis 1997; 3: S102–S109
   PubMedGoogle Scholar
• Odds FC. Biotyping of medically important fungi. Curr Top Med Mycol 1985; 1: 155–171
   PubMedGoogle Scholar