References
- Kobayashi T, Dynlacht BD. Regulating the transition from centriole to basal body. J Cell Biol 2011; 193:435-44; PMID:21536747; http://dx.doi.org/10.1083/jcb.201101005
- Rooman I, Real FX. Pancreatic ductal adenocarcinoma and acinar cells: a matter of differentiation and development?. Gut 2012; 61:449-58; PMID:21730103; http://dx.doi.org/10.1136/gut.2010.235804
- Seeley ES, Carrière C, Goetze T, Longnecker DS, Korc M. Pancreatic cancer and precursor pancreatic intraepithelial neoplasia lesions are devoid of primary cilia. Cancer Res 2009; 69:422-30; PMID:19147554; http://dx.doi.org/10.1158/0008-5472.CAN-08-1290
- Kobayashi T, Nakazono K, Tokuda M, Mashima Y, Dynlacht BD, Itoh H. HDAC2 promotes loss of primary cilia in pancreatic ductal adenocarcinoma. EMBO Rep 2017; 18:334-43; PMID:28028031; http://dx.doi.org/10.15252/embr.201541922
- Pugacheva EN, Jablonski SA, Hartman TR, Henske EP, Golemis EA. HEF1-dependent Aurora A activation induces disassembly of the primary cilium. Cell 2007; 129:1351-63; PMID:17604723; http://dx.doi.org/10.1016/j.cell.2007.04.035
- Cervantes S, Lau J, Cano DA, Borromeo-Austin C, Hebrok M. Primary cilia regulate Gli/Hedgehog activation in pancreas. Proc Natl Acad Sci U S A 2010; 107:10109-14; PMID:20479231; http://dx.doi.org/10.1073/pnas.0909900107
- Yauch RL, Gould SE, Scales SJ, Tang T, Tian H, Ahn CP, Marshall D, Fu L, Januario T, Kallop D, et al. A paracrine requirement for hedgehog signalling in cancer. Nature 2008; 455:406-10; PMID:18754008; http://dx.doi.org/10.1038/nature07275