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Editorials: Cell Cycle Features

TIRR and 53BP1- partners in arms

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Pages 1235-1236 | Received 27 Apr 2017, Accepted 09 May 2017, Published online: 23 Jun 2017

References

  • Zimmermann M, de Lange T. 53BP1: pro choice in DNA repair Trends Cell Biol 2014; 24:108-17; PMID:24094932; https://doi.org/10.1016/j.tcb.2013.09.003
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  • Drane P, Brault ME, Cui G, Meghani K, Chaubey S, Detappe A, Parnandi N, He Y, Zheng XF, Botuyan MV, Kalousi A, Yewdell WT, Munch C, Harper JW, Chaudhuri J, Soutoglou E, Mer G, Chowdhury D. TIRR regulates 53BP1 by masking its histone methyl-lysine binding function Nature 2017; 543:211-6; PMID:28241136; https://doi.org/10.1038/nature21358
  • Zhang A, Peng B, Huang P, Chen J, Gong Z. The p53-binding protein 1-Tudor-interacting repair regulator complex participates in the DNA damage response J Biol Chem 2017; 292, 6461-67; PMID:28213517; https://doi.org/10.1074/jbc.M117.777474
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  • Zong D, Callen E, Pegoraro G, Lukas C, Lukas J, Nussenzweig A. Ectopic expression of RNF168 and 53BP1 increases mutagenic but not physiological non-homologous end joining Nucleic Acids Res 2015; 43:4950-61; PMID:25916843; https://doi.org/10.1093/nar/gkv336

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