Oh, SNAP! How enteroviruses redirect autophagic traffic away from degradation

Abigail K. CoronaDepartment of Microbiology and Immunology, University of Maryland School of Medicine, Baltimore, MD, USAView further author information

Yasir MohamudCenter for Heart Lung Innovation, St. Paul’s Hospital and Department of Pathology and Laboratory Medicine, University of British Columbia, Vancouver, BC, CanadaView further author information

William T. JacksonDepartment of Microbiology and Immunology, University of Maryland School of Medicine, Baltimore, MD, USACorrespondence[email protected]

http://orcid.org/0000-0002-9832-0584 View further author information

Honglin LuoCenter for Heart Lung Innovation, St. Paul’s Hospital and Department of Pathology and Laboratory Medicine, University of British Columbia, Vancouver, BC, CanadaView further author information

References

Mohamud Y, Shi J, Qu J, et al. Enteroviral infection inhibits autophagic flux via disruption of the SNARE complex to enhance viral replication. Cell Rep. 2018;22(12):3292–3303. PMID:29562184.
Web of Science ®Google Scholar
Corona AK, Saulsbery HM, Corona Velazquez AF, et al. Enteroviruses remodel autophagic trafficking through regulation of host SNARE proteins to promote virus replication and cell exit. Cell Rep. 2018;22(12):3304–3314. PMID:29562185.
Web of Science ®Google Scholar

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Oh, SNAP! How enteroviruses redirect autophagic traffic away from degradation

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