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Autophagy Volume 14, 2018 - Issue 12
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Commentary

BECN1 is a new driver of ferroptosis

Rui KangThe Third Affiliated Hospital, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China;Department of Surgery, University of Pittsburgh, Pittsburgh, PA, USA;Department of Surgery, The University of Texas Southwestern Medical Center, Dallas, TX, USACorrespondence[email protected] [email protected]
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,
Shan ZhuThe Third Affiliated Hospital, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, ChinaView further author information
,
Herbert J. ZehDepartment of Surgery, University of Pittsburgh, Pittsburgh, PA, USA;Department of Surgery, The University of Texas Southwestern Medical Center, Dallas, TX, USAView further author information
,
Daniel J. KlionskyLife Sciences Institute and Department of Molecular, Cellular and Developmental Biology, University of Michigan, Ann Arbor, MI, USAORCID Iconhttp://orcid.org/0000-0002-7828-8118View further author information
ORCID Icon &
Daolin TangThe Third Affiliated Hospital, School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, China;Department of Surgery, University of Pittsburgh, Pittsburgh, PA, USA;Department of Surgery, The University of Texas Southwestern Medical Center, Dallas, TX, USACorrespondence[email protected] [email protected]
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Pages 2173-2175 | Received 11 Aug 2018, Accepted 15 Aug 2018, Published online: 06 Sep 2018

References

  • Song X, Zhu S, Chen P, et al. AMPK-Mediated BECN1 phosphorylation promotes ferroptosis by directly blocking system X(c)(-) activity. Curr Biol. 2018 Aug 6;28(15):2388–2399.e5. Epub 2018 Jul 26. PubMed PMID: 30057310.

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