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Autophagy Volume 16, 2020 - Issue 9
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Autophagic punctum

Hepatic peroxisomal β-oxidation suppresses lipophagy via RPTOR acetylation and MTOR activation

Anyuan HeDivision of Endocrinology, Metabolism and Lipid Research, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USACorrespondence[email protected]
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,
John M. DeanDivision of Endocrinology, Metabolism and Lipid Research, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USAView further author information
,
Dongliang LuDivision of Endocrinology, Metabolism and Lipid Research, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USAView further author information
,
Yali ChenDivision of Endocrinology, Metabolism and Lipid Research, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USAView further author information
&
Irfan J. LodhiDivision of Endocrinology, Metabolism and Lipid Research, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USACorrespondence[email protected]
ORCID Iconhttps://orcid.org/0000-0002-6246-9862View further author information
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Pages 1727-1728 | Received 21 Jun 2020, Accepted 09 Jul 2020, Published online: 27 Jul 2020

Reference

  • He A, Chen X, Tan M, et al. Acetyl-CoA derived from hepatic peroxisomal β-oxidation inhibits autophagy and promotes steatosis via MTORC1 activation. Mol Cell. 2020 Jul 2;79(1):30–42.e4.

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