References
- Sun H. The interaction between pathogens and the host coagulation system. Physiology. 2006;21(4):281–288.
- Chapin JC, Hajjar KA. Fibrinolysis and the control of blood coagulation. Blood Rev. 2015;29(1):17–24. doi:https://doi.org/10.1016/j.blre.2014.09.003.
- Prabhudesai A, Shetty S, Ghosh K, et al. Dysfunctional fibrinolysis and cerebral venous thrombosis. YBCMD blood cells. Mol Dis. 2017;65:51–55.
- Jin NZ, Gopinath SCB. Potential blood clotting factors and anticoagulants. Biomed Pharmacother. 2016;84:356–365.
- Ong CC, Gopinath SCB, Rebecca LWX, et al. Diagnosing human blood clotting deficiency. Int J Biol Macromol. 2018;116:765–773. doi:https://doi.org/10.1016/j.ijbiomac.2018.05.084.
- Batty P, Smith JG. Haemostasis. Surgery (Oxford). 2010;28(11):530–535. doi:https://doi.org/10.1016/j.mpsur.2010.08.008.
- Lijnen HR. Elements of the fibrinolytic system. Ann N Y Acad Sci. 2001;936(1):226–236.
- Wiman B, Collen D. Molecular mechanism of physiological fibrinolysis. Nature. 1978;272(5653):549–550.
- Collen D, Lijnen HR. Basic and clinical aspects of fibrinolysis and thrombolysis. Blood. 1991;78(12):3114–3124. doi:https://doi.org/10.1182/blood.V78.12.3114.bloodjournal78123114.
- Franchini M, Mannucci PM. Primary hyperfibrinolysis: facts and fancies. Thromb Res. 2018;166:71–75.
- Collen D. The plasminogen (fibrinolytic) system. Thromb Haemost. 1999;82(08):837–870.
- Cesarman-Maus G, Hajjar KA. Molecular mechanisms of fibrinolysis. Br J Haematol. 2005;129(3):307–321. doi:https://doi.org/10.1111/j.1365-2141.2005.05444.x.
- Yang R, Xie T, Yang H, et al. Historical trends of organochlorine pesticides (OCPs) recorded in sediments across the Tibetan plateau. Environ Geochem Health. 2018;40(1):303–312.
- Wang Z, Liu F, Ye S, et al. Plasma proteome profiling of high-altitude polycythemia using TMT-based quantitative proteomics approach. J Proteomics. 2019;194:60–69. doi:https://doi.org/10.1016/j.jprot.2018.12.031.
- Du X, Zhang R, Ye S, et al. Alterations of human plasma proteome profile on adaptation to high-altitude hypobaric hypoxia. J Proteome Res. 2019;18(5):2021–2031. doi:https://doi.org/10.1021/acs.jproteome.8b00911.
- Parati G, Agostoni P, Basnyat B, et al. Clinical recommendations for high altitude exposure of individuals with pre-existing cardiovascular conditions: A joint statement by the European Society of Cardiology, the Council on Hypertension of the European Society of Cardiology, the European Society of Hypertension, the International Society of Mountain Medicine, the Italian Society of Hypertension and the Italian Society of Mountain Medicine. Eur Heart J. 2018;39(17):1546–1554.
- West JB. English translation of “Nomenclature, classification, and diagnostic criteria of high altitude disease in China”. High Alt Med Biol. 2010;11(2):169–172. doi:https://doi.org/10.1089/ham.2010.1014.
- Li C, Li X, Liu J, et al. Investigation of the differences between the Tibetan and Han populations in the hemoglobin-oxygen affinity of red blood cells and in the adaptation to high-altitude environments. Hematology. 2018;23(5):309–313. doi:https://doi.org/10.1080/10245332.2017.1396046.
- Zhong R, Liu H, Wang H, et al. Adaption to high altitude: An evaluation of the storage quality of suspended Red blood cells prepared from the whole blood of tibetan plateau migrants. PLoS One. 2015;10(12):e0144201, doi:https://doi.org/10.1371/journal.pone.0144201.
- Liu C, Liu B, Zhang EL, et al. Elevated pentose phosphate pathway is involved in the recovery of hypoxiainduced erythrocytosis. Mol Med Rep. 2017;16(6):9441–9448. doi:https://doi.org/10.3892/mmr.2017.7801.
- Damodar D, Donnally CJ, Sheu JI, et al. A higher altitude is an independent risk factor for venous thromboembolisms after total Hip arthroplasty. J Arthroplasty. 2018;33(8):2627–2630.
- Rocke AS, Paterson GG, Barber MT, et al. Thromboelastometry and platelet function during acclimatization to high altitude. Thromb Haemost. 2018;118(1):063–071.
- Wheatley K, Creed M, Mellor A. Haematological changes at altitude. J R Army Med Corps. 2011;157(1):38–42.
- Gupta N, Ashraf MZ. Exposure to high altitude: A risk factor for venous thromboembolism? Semin Thromb Hemost. 2012;38(2):156–163.
- Wang Z, Liu H, Dou M, et al. The quality changes in fresh frozen plasma of the blood donors at high altitude. PLoS One. 2017;12(4):e0176390. doi:https://doi.org/10.1371/journal.pone.0176390.
- Zhang R, Yu X, Shen Y, et al. Correlation between RBC changes and coagulation parameters in high altitude population. Hematology. 2019;24(1):325–330. doi:https://doi.org/10.1080/16078454.2019.1568658.
- Wang Z, Dou M, Du X, et al. Influences of ABO blood group, age and gender on plasma coagulation factor VIII, fibrinogen, von Willebrand factor and ADAMTS13 levels in a Chinese population. PeerJ. 2017;5:e3156. doi:https://doi.org/10.7717/peerj.3156.
- Kotwal J, Apte CV, Kotwal A, et al. High altitude: a hypercoagulable state: results of a prospective cohort study. Thromb Res. 2007;120(3):391–397.
- Johari V, Loke C. Brief overview of the coagulation cascade. Disease A Month. 2012;58(8):421–423.
- Khalafallah A, Morse M, Stewart P, et al. Evaluation of the innovance d-dimer assay for the diagnosis of disseminated intravascular coagulopathy in different clinical settings. Clin Appl Thromb Hemost. 2014;20(1):91–97.
- van der Hulle T, Tan M, den Exter PL, et al. selective D-dimer testing for the diagnosis of acute deep vein thrombosis: a validation study. J Thromb Haemost Haemost. 2013;11(12):2184–2186.
- Van der Hulle T, Den Exter PL, Erkens PGM, et al. Variable D-dimer thresholds for diagnosis of clinically suspected acute pulmonary embolism. J Thromb Haemostasis. 2013;11(11):1986–1992.
- Crosby A, Talbot NP, Harrison P, et al. Relation between acute hypoxia and activation of coagulation in human beings. Lancet. 2003;361(9376):2207–2208.
- León-Velarde F, Maggiorini M, Reeves JT, et al. Consensus statement on chronic and subacute high altitude diseases. High Altitude Med Biol. 2005;6(2):147–157.