Nutritional status and risk of amyotrophic lateral sclerosis in Japan

References

• Statistics and Information Department, Ministers Secretariat. Vital statistics of Japan 1995–2005. The Ministry of Health, Labor and Welfare, Tokyo
   Google Scholar
• Strickland D., Smith S. A., Dolliff G., Goldman L., Roelofs R. I. Physical activity, trauma, and ALS: a case‐control study. Acta Neurol Scand 1996; 94: 45–50
   PubMed Web of Science ®Google Scholar
• Longstreth W. T., McGuire V., Koepsell T. D., Wang Y., van Belle G. Risk of amyotrophic lateral sclerosis and history of physical activity: a population‐based case‐control study. Arch Neurol 1998; 55: 201–6
   PubMedGoogle Scholar
• Veldink J. H., Kalmijn S., Groeneveld G. J., Titulaer M. J., Wokke J. H., van den Berg L. H. Physical activity and the association with sporadic ALS. Neurology 2005; 64: 241–5
   PubMed Web of Science ®Google Scholar
• Gresham L. S., Molgaard C. A., Golbeck A. L., Smith R. Amyotrophic lateral sclerosis and history of skeletal fracture: a case‐control study. Neurology 1987; 37: 717–9
   PubMed Web of Science ®Google Scholar
• Deapen D. M., Henderson B. E. A case‐control study of amyotrophic lateral sclerosis. Am J Epidemiol 1986; 123: 790–9
   PubMed Web of Science ®Google Scholar
• McGuire V., Longstreth W. T., Nelson L. M., Koepsell T. D., Checkoway H., Morgan M. S., van Belle G. Occupational exposures and amyotrophic lateral sclerosis. A population‐based case‐control study. Am J Epidemiol 1997; 145: 1076–88
   PubMed Web of Science ®Google Scholar
• Bergomi M., Vinceti M., Nacci G., Pietrini V., Bratter P., Alber D., et al. Environmental exposure to trace elements and risk of amyotrophic lateral sclerosis: a population‐based case‐control study. Environ Res 2002; 89: 116–23
   PubMed Web of Science ®Google Scholar
• Longnecker M. P., Kamel F., Umbach D. M., Munsat T. L., Shefner J. M., Lansdell L. W., et al. Dietary intake of calcium, magnesium and antioxidants in relation to risk of amyotrophic lateral sclerosis. Neuroepidemiology 2000; 19: 210–6
   PubMed Web of Science ®Google Scholar
• Yasui M., Ota K., Yoshida M. Effects of low calcium and magnesium dietary intake on the central nervous system tissues of rats and calcium‐magnesium related disorders in the amyotrophic lateral sclerosis focus in the Kii Peninsula of Japan. Magnes Res 1997; 10: 39–50
   PubMed Web of Science ®Google Scholar
• Ascherio A., Weisskopf M. G., O'Reilly E. J., Jacobs E. J., McCullough M. L., Calle E. E., et al. Vitamin E intake and risk of amyotrophic lateral sclerosis. Ann Neurol 2005; 57: 104–10
   PubMed Web of Science ®Google Scholar
• Galbussera A., Tremolizzo L., Brighina L., Testa D., Lovati R., Ferrarese C., et al. Vitamin E intake and quality of life in amyotrophic lateral sclerosis patients: a follow‐up case series study. Neurol Sci 2006; 27: 190–3
   PubMed Web of Science ®Google Scholar
• 1998, World Federation of Neurology Research Group on Motor Neuron Diseases: El Escorial Revisited: Revised Criteria for the Diagnosis of Amyotrophic Lateral Sclerosis. A consensus conference held at Airlie House, Warrenton, Virginia April 2–4
   Google Scholar
• Wakai K., Egami I., Kato K., Lin Y., Kawamura T., Tamakoshi A., et al. A simple food frequency questionnaire for Japanese diet – Part I. Development of the questionnaire, and reproducibility and validity for food groups. J Epidemiol 1999; 216–26, 9
   PubMedGoogle Scholar
• Egami I., Wakai K., Kato K., Lin Y., Kawamura T., Tamakoshi A., et al. A simple food frequency questionnaire for Japanese diet – Part II. Development of the questionnaire, and reproducibility and validity for food groups. J Epidemiol 1999; 227–34, 9
   PubMedGoogle Scholar
• Drewnowski A., Hann C. Food preferences and reported frequencies of food consumption as predictors of current diet in young women. Am J Clin Nutr 1999; 70: 28–36
   PubMed Web of Science ®Google Scholar
• Friedman M., Rosenman R. H. Type A behavior and your heart. Knopf, New York 1974
   Google Scholar
• Feskens E. J., Bowles C. H., Kromhout D. Carbohydrate intake and body mass index in relation to the risk of glucose intolerance in an elderly population. Am J Clin Nutr 1991; 54: 136–40
   PubMed Web of Science ®Google Scholar
• Clapp J. F 3rd. Effect of dietary carbohydrate on the glucose and insulin response to mixed caloric intake and exercise in both non‐pregnant and pregnant women. Diabetes Care 1998; 21 (Suppl 2): B107–12
   PubMedGoogle Scholar
• Kang B. P. S., Frencher S., Reddy V., Kessler A., Malhorta A., Meggs L. G. High glucose promotes messengial cell apoptosis by oxidant‐dependent mechanism. Am J Physiol Renal Physiol 2003; 284: F455–66
   PubMed Web of Science ®Google Scholar
• Allen D. A., Harwood S. M., Varagunam M., Raaftery M. J., Yaqoob M. M. 2003, High glucose‐induced oxidative stress causes apoptosis in proximal tubular epithelial cells and is mediated by multiple caspases. The FASEB Journal express doi:10.1096/fj.02‐0130fje. Published online March 28
   Google Scholar
• Allen D. A., Yaqoob M. M., Harwood S. M. Mechanism of high glucose‐induced apoptosis and its relationship to diabetic complications. J Nutr Biochem 2005; 16: 705–13
   PubMed Web of Science ®Google Scholar
• Sheu M. L., Ho F. M., Yang R. S., Chao K. F., Lin W. W., Lin‐Shiau S. Y., et al. High glucose induces human endothelial cell apoptosis through a phosphoinositide 3‐kinase‐regulated cyclooxygenase‐2 pathway. Arterioscler Thromb Vasc Biol 2005; 25: 539–45
   PubMed Web of Science ®Google Scholar
• Murai A., Miyahara T., Tanaka T., Kaneko T., Sako Y., Kameyama M. Abnormalities of lipoprotein and carbohydrate metabolism in degenerative diseases of the nervous system: motor neuron disease and spinocerebellar degeneration. Tohoku J Exp Med 1983; 139: 365–76
   PubMedGoogle Scholar
• Russo V. C., Kobayashi K., Najdovska S., Baker N. L., Werther G. A. Neuronal protection from glucose deprivation via modulation of glucose transport and inhibition of apoptosis: a role for the insulin‐like growth factor system. Brain Res 2004; 1009: 40–53
   PubMed Web of Science ®Google Scholar
• Luyer M. D., Greve J. W., Hadfoune M., Jacobs J. A., Dejong C. H., Buurman W. A. Nutritional stimulation of cholecystokinin receptors inhibits inflammation via the vagus nerve. J Exp Med 2005; 202: 1023–9
   PubMed Web of Science ®Google Scholar
• Perez J., Ware M. A., Chevalier S., Gougeon R., Bennett G. J., Shir Y. Dietary fat and protein interact in suppressing neuropathic pain‐related disorders following a partial sciatic ligation injury in rats. Pain 2004; 111: 297–305
   PubMedGoogle Scholar
• Wang Z. J., Li G. M., Nie B. M., Lu Y., Yin M. Neuroprotective effect of stearic acid against oxidative stress via phosphatidylinositol 3‐kinase pathway. Chem Biol Interact 2006; 160: 80–7
   PubMedGoogle Scholar
• Velasco A., Tabernero A., Medina J. M. Role of oleic acid as a neurotrophic factor is supported in vivo by the expression of GAP‐43 subsequent to the activation of SREBP‐1 and the up‐regulation of stearoyl‐CoA desaturase during post‐natal development of the brain. Brain Res 2003; 977: 103–11
   PubMed Web of Science ®Google Scholar
• Fressinaud C., Vallat J. M., Rigaud M., Leboutet M. J. Analysis of unsaturated fatty acids of endoneurium and perineurium from normal and degenerating rat sciatic nerve. Morphological correlations. J Neurol Sci 1987; 81: 85–92
   PubMedGoogle Scholar
• Bourre J. M., Dumont O., Piciotti M., Pascal G., Durand G. Control of brain fatty acids. Ups J Med Sci Supple 1990; 48: 109–31
   PubMedGoogle Scholar
• Beeharry N., Lowe J. E., Hernandez A. R., Chambers J. A., Fucassi F., Cragg P. J., et al. Linoleic acid and antioxidants protect against DNA damage and apoptosis induced by palmitic acid. Mutat Res 2003; 530: 27–33
   PubMed Web of Science ®Google Scholar
• Okamoto K., Ohno Y., Horisawa R., Wakai K., Tamakoshi A., Kawamura T. Agreement between self‐ and partner‐reports obtained by a self‐administered questionnaire: medical and lifestyle information. J Epidemiol 1999; 183–9, 9
   PubMedGoogle Scholar
• Marshall J., Priore R., Haughey B., Rzepka T., Graham S. Spouse‐subject interview and the reliability of diet studies. Am J Epidemiol 1980; 112: 675–83
   PubMed Web of Science ®Google Scholar